The association between MTHFR polymorphisms and cervical cancer risk: a system review and meta analysis

[1] Echelman D(2012)Management of cervical precancers: a global perspective Hematol Oncol Clin North Am 26 31-44

[2] Feldman S(2003)Epidemiologic classification of human papillomavirus types associated with cervical cancer N Engl J Med 348 518-527

[3] Munoz N(1999)Human papillomavirus is a necessary cause of invasive cervical cancer worldwide J Pathol 189 12-19

[4] Bosch FX(2000)Smoking, diet, pregnancy and oral contraceptive use as risk factors for cervical intra-epithelial neoplasia in relation to human papillomavirus infection Br J Cancer 82 1332-1338

[5] de Sanjose S(2000)Viral load of human papilloma virus 16 as a determinant for development of cervical carcinoma in situ: a nested case-control study Lancet 355 2189-2193

[6] Herrero R(1999)Polymorphisms of methylenetetrahydrofolate reductase and other enzymes: metabolic significance, risks and impact on folate requirement J Nutr 129 919-922

[7] Castellsague X(1988)Intermediate homocysteinemia: a thermolabile variant of methylenetetrahydrofolate reductase Am J Hum Genet 43 414-421

[8] Shah KV(1995)A candidate genetic risk factor for vascular disease: a common mutation in methylenetetrahydrofolate reductase Nat Genet 10 111-113

[9] Snijders PJ(1994)Human methylenetetrahydrofolate reductase: isolation of cDNA mapping and mutation identification Nat Genet 7 551-261

[10] Meijer CJ(2014)Study on the relationship between the MTHFR polymorphism, the level of the folic acid and the cervical cancer susceptibility Sichuan Da Xue Xue Bao Yi Xue Ban 45 258-72

[1] Echelman D(2012)Management of cervical precancers: a global perspective Hematol Oncol Clin North Am 26 31-44

[2] Feldman S(2003)Epidemiologic classification of human papillomavirus types associated with cervical cancer N Engl J Med 348 518-527

[3] Munoz N(1999)Human papillomavirus is a necessary cause of invasive cervical cancer worldwide J Pathol 189 12-19

[4] Bosch FX(2000)Smoking, diet, pregnancy and oral contraceptive use as risk factors for cervical intra-epithelial neoplasia in relation to human papillomavirus infection Br J Cancer 82 1332-1338

[5] de Sanjose S(2000)Viral load of human papilloma virus 16 as a determinant for development of cervical carcinoma in situ: a nested case-control study Lancet 355 2189-2193

[6] Herrero R(1999)Polymorphisms of methylenetetrahydrofolate reductase and other enzymes: metabolic significance, risks and impact on folate requirement J Nutr 129 919-922

[7] Castellsague X(1988)Intermediate homocysteinemia: a thermolabile variant of methylenetetrahydrofolate reductase Am J Hum Genet 43 414-421

[8] Shah KV(1995)A candidate genetic risk factor for vascular disease: a common mutation in methylenetetrahydrofolate reductase Nat Genet 10 111-113

[9] Snijders PJ(1994)Human methylenetetrahydrofolate reductase: isolation of cDNA mapping and mutation identification Nat Genet 7 551-261

[10] Meijer CJ(2014)Study on the relationship between the MTHFR polymorphism, the level of the folic acid and the cervical cancer susceptibility Sichuan Da Xue Xue Bao Yi Xue Ban 45 258-72