Association of apolipoprotein A1 -75 G/A polymorphism with susceptibility to the development of acute lung injury after cardiopulmonary bypass surgery

[1] Hammermeister KE(1990)Identification of patients at greatest risk for developing major complications at cardiac surgery Circulation 82 IV380-IV389

[2] Burchfiel C(2007)Neutrophil elastase inhibitor, sivelestat, attenuates acute lung injury after cardiopulmonary bypass in the rabbit endotoxemia model Ann Thorac Surg 83 153-160

[3] Johnson R(1985)Acute lung injury during cardiopulmonary bypass, the role of neutrophil sequestration and lipid peroxidation Trans Am Soc Artif Intern Organs 31 604-609

[4] Grover FL(1990)Leukocyte depletion ameliorates free radical-mediated lung injury after cardiopulmonary bypass J Thorac Cardiovasc Surg 99 873-877

[5] Wakayama F(1999)Lung injury and acute respiratory distress syndrome after cardiopulmonary bypass Ann Thorac Surg 68 1107-1115

[6] Fukuda I(2003)Future research directions in acute lung injury: summary of a National Heart, Lung, and Blood Institute working group Am J Respir Crit Care Med 167 1027-1035

[7] Suzuki Y(2007)ApolipoproteinA1–75 G/A (M1-) polymorphism and lipoprotein(a); anti- vs. pro-Atherogenic properties Lipids Health Dis 6 19-264

[8] Kondo N(1994)Guanidine to adenine (G/A) substitution in the promoter region of the apolipoprotein AI gene is associated with elevated serum apolipoprotein AI levels in Chinese non-smokers Genet Epidemiol 11 255-824

[9] Westaby S(1994)The American-European Consensus Conference on ARDS. Definitions, mechanisms, relevant outcomes, and clinical trial coordination Am J Respir Crit Care Med 149 818-1539

[10] Fleming J(2009)Genetic polymorphisms associated with acute lung injury Pharmacogenomics 10 1527-249

[1] Hammermeister KE(1990)Identification of patients at greatest risk for developing major complications at cardiac surgery Circulation 82 IV380-IV389

[2] Burchfiel C(2007)Neutrophil elastase inhibitor, sivelestat, attenuates acute lung injury after cardiopulmonary bypass in the rabbit endotoxemia model Ann Thorac Surg 83 153-160

[3] Johnson R(1985)Acute lung injury during cardiopulmonary bypass, the role of neutrophil sequestration and lipid peroxidation Trans Am Soc Artif Intern Organs 31 604-609

[4] Grover FL(1990)Leukocyte depletion ameliorates free radical-mediated lung injury after cardiopulmonary bypass J Thorac Cardiovasc Surg 99 873-877

[5] Wakayama F(1999)Lung injury and acute respiratory distress syndrome after cardiopulmonary bypass Ann Thorac Surg 68 1107-1115

[6] Fukuda I(2003)Future research directions in acute lung injury: summary of a National Heart, Lung, and Blood Institute working group Am J Respir Crit Care Med 167 1027-1035

[7] Suzuki Y(2007)ApolipoproteinA1–75 G/A (M1-) polymorphism and lipoprotein(a); anti- vs. pro-Atherogenic properties Lipids Health Dis 6 19-264

[8] Kondo N(1994)Guanidine to adenine (G/A) substitution in the promoter region of the apolipoprotein AI gene is associated with elevated serum apolipoprotein AI levels in Chinese non-smokers Genet Epidemiol 11 255-824

[9] Westaby S(1994)The American-European Consensus Conference on ARDS. Definitions, mechanisms, relevant outcomes, and clinical trial coordination Am J Respir Crit Care Med 149 818-1539

[10] Fleming J(2009)Genetic polymorphisms associated with acute lung injury Pharmacogenomics 10 1527-249