Effects of lamotrigine on conditiioned learning after global cerebral ischemia in rabbits

[1] Scholz W., Selective neuronal necrosis and its topistic patterns in hypoxemia, J. Neuropathol. Exp. Neurol., 12, pp. 249-261, (1953)

[2] Benveniste H., Drejer J., Schousboe A., Diemer N.H., Elevation of the extracellular concentrations of glutamate and aspartate in rat hippocampus during transient cerebral ischemia monitored by intracerebral microdialysis, J. Neurochem., 43, pp. 1369-1374, (1984)

[3] Thompson R., The neurobiology of learning and memory, Science, 233, pp. 941-947, (1986)

[4] Moyer J.R., Deyo R.A., Disterhoft J.F., Hippocampectomy disrupts trace eye-blink conditioning in rabbits, Behav. Neurosci., 104, pp. 243-252, (1990)

[5] Woodruff-Pak D.S., Lavond D.G., Thompson R.F., Trace conditioning: Abolished by cerebellar nuclear lesions but not lateral cerebellar cortex aspirations, Brain Res., 348, pp. 249-260, (1985)

[6] Kwon J.Y., Bacher A., Deyo D.J., Grafe M.R., Disterhoft J.F., Uchida T., Zornow M.H., Effects of hypothermia and lamotrigine on trace-conditioned learning after global cerebral ischemia in rabbits, Exp. Neurol., 159, pp. 105-113, (1999)

[7] Harvey J.A., Gormezano I., Cool-Hauser V.A., Relationship between heterosynaptic reflex facilitation and acquisition of the nictitating membrane response in control and scopolamine-injected rabbits, J. Neurosci., 5, pp. 596-602, (1985)

[8] Graves C.A., Solomon P.R., Age-related disruption of trace but not delay classical conditioning of the rabbit's nictitating membrane response, Behav. Neurosci., 99, pp. 88-96, (1985)

[9] Pulsinelli W.A., Brierly J.B., Plum F., Temporal profile of neuronal damage in a model of transient forebrain ischemia, Ann. Neurol., 11, pp. 491-498, (1982)

[10] Baker A.J., Zornow M.H., Grafe M.R., Scheller M.S., Skilling S.R., Smullin D.H., Larson A.A., Hypothermia prevents ischemia-induced increases in hippocampal glycine concentrations in rabbits, Stroke, 22, pp. 666-673, (1991)

[1] Scholz W., Selective neuronal necrosis and its topistic patterns in hypoxemia, J. Neuropathol. Exp. Neurol., 12, pp. 249-261, (1953)

[2] Benveniste H., Drejer J., Schousboe A., Diemer N.H., Elevation of the extracellular concentrations of glutamate and aspartate in rat hippocampus during transient cerebral ischemia monitored by intracerebral microdialysis, J. Neurochem., 43, pp. 1369-1374, (1984)

[3] Thompson R., The neurobiology of learning and memory, Science, 233, pp. 941-947, (1986)

[4] Moyer J.R., Deyo R.A., Disterhoft J.F., Hippocampectomy disrupts trace eye-blink conditioning in rabbits, Behav. Neurosci., 104, pp. 243-252, (1990)

[5] Woodruff-Pak D.S., Lavond D.G., Thompson R.F., Trace conditioning: Abolished by cerebellar nuclear lesions but not lateral cerebellar cortex aspirations, Brain Res., 348, pp. 249-260, (1985)

[6] Kwon J.Y., Bacher A., Deyo D.J., Grafe M.R., Disterhoft J.F., Uchida T., Zornow M.H., Effects of hypothermia and lamotrigine on trace-conditioned learning after global cerebral ischemia in rabbits, Exp. Neurol., 159, pp. 105-113, (1999)

[7] Harvey J.A., Gormezano I., Cool-Hauser V.A., Relationship between heterosynaptic reflex facilitation and acquisition of the nictitating membrane response in control and scopolamine-injected rabbits, J. Neurosci., 5, pp. 596-602, (1985)

[8] Graves C.A., Solomon P.R., Age-related disruption of trace but not delay classical conditioning of the rabbit's nictitating membrane response, Behav. Neurosci., 99, pp. 88-96, (1985)

[9] Pulsinelli W.A., Brierly J.B., Plum F., Temporal profile of neuronal damage in a model of transient forebrain ischemia, Ann. Neurol., 11, pp. 491-498, (1982)

[10] Baker A.J., Zornow M.H., Grafe M.R., Scheller M.S., Skilling S.R., Smullin D.H., Larson A.A., Hypothermia prevents ischemia-induced increases in hippocampal glycine concentrations in rabbits, Stroke, 22, pp. 666-673, (1991)