Long-term potentiation of glutamatergic transmission and epileptiform activity induced by transient episodes of anoxia in slices of rats hippocampus field CA1

[1] Sem'yanov A.V., Kalemenev S.V., Godukhin O.V., A kindling-like state induced by periodic increases in the extracellular K⁺ concentration in field CA1 in rat hippocampus slices, Ros. Fiziol. Zh. im. I. M. Sechenova, 83, 18, pp. 14-23, (1997)

[2] Sem'yanov A.V., Godukhin O.V., Prolonged decreases in the threshold for evoked epileptiform discharges in slices of rat hippocampus field CA1 induced by periodic removal of Mg²⁺, Ros. Fiziol. Zh. im. I. M. Sechenova, 84, 12, pp. 1438-1442, (1998)

[3] Applegate C.D., Jensen F.E., Burchfield J.L., Lombroso C., The effects of neonatal hypoxia on kindled seizure development and electroconvulsive shock profiles, Epilepsia, 37, 8, pp. 723-727, (1996)

[4] Chiba S., Long-term effect of postnatal hypoxia on the seizure susceptibility in rats, Life Sci., 37, 17, pp. 1597-1604, (1985)

[5] Hammond C., Crepel V., Gozlan H., Ben-Ari Y., Anoxic LTP sheds light on the multiple facts of NMDA receptors, TINS, 17, 11, pp. 497-503, (1994)

[6] Obrenovich T.P., Urenjak J., Zikha E., Evidence disputing the link between seizure activity and high extracellular glutamate, J. Neurochem., 66, pp. 2446-2454, (1996)

[7] Jensen F.E., Wang C., Stafstrom C.E., Liu Z., Geary C., Stevens M.C., Acute and chronic increases in excitability in rat hippocampal slices after perinatal hypoxia in vivo, J. Neurophys., 79, 1, pp. 73-81, (1998)

[8] Romijen H.J., Voskuyl R.A., Coenen A.M., Hypoxic-ischemic encephalopathy sustained in early postnatal life may result in permanent epileptic activity and an altered cortical convulsive threshold in rat, Epilepsy Res., 17, 1, pp. 31-42, (1994)

[1] Sem'yanov A.V., Kalemenev S.V., Godukhin O.V., A kindling-like state induced by periodic increases in the extracellular K⁺ concentration in field CA1 in rat hippocampus slices, Ros. Fiziol. Zh. im. I. M. Sechenova, 83, 18, pp. 14-23, (1997)

[2] Sem'yanov A.V., Godukhin O.V., Prolonged decreases in the threshold for evoked epileptiform discharges in slices of rat hippocampus field CA1 induced by periodic removal of Mg²⁺, Ros. Fiziol. Zh. im. I. M. Sechenova, 84, 12, pp. 1438-1442, (1998)

[3] Applegate C.D., Jensen F.E., Burchfield J.L., Lombroso C., The effects of neonatal hypoxia on kindled seizure development and electroconvulsive shock profiles, Epilepsia, 37, 8, pp. 723-727, (1996)

[4] Chiba S., Long-term effect of postnatal hypoxia on the seizure susceptibility in rats, Life Sci., 37, 17, pp. 1597-1604, (1985)

[5] Hammond C., Crepel V., Gozlan H., Ben-Ari Y., Anoxic LTP sheds light on the multiple facts of NMDA receptors, TINS, 17, 11, pp. 497-503, (1994)

[6] Obrenovich T.P., Urenjak J., Zikha E., Evidence disputing the link between seizure activity and high extracellular glutamate, J. Neurochem., 66, pp. 2446-2454, (1996)

[7] Jensen F.E., Wang C., Stafstrom C.E., Liu Z., Geary C., Stevens M.C., Acute and chronic increases in excitability in rat hippocampal slices after perinatal hypoxia in vivo, J. Neurophys., 79, 1, pp. 73-81, (1998)

[8] Romijen H.J., Voskuyl R.A., Coenen A.M., Hypoxic-ischemic encephalopathy sustained in early postnatal life may result in permanent epileptic activity and an altered cortical convulsive threshold in rat, Epilepsy Res., 17, 1, pp. 31-42, (1994)