The Henna pigment Lawsone activates the Aryl Hydrocarbon Receptor and impacts skin homeostasis

被引:0
作者
Laura Lozza
Pedro Moura-Alves
Teresa Domaszewska
Carolina Lage Crespo
Ioana Streata
Annika Kreuchwig
Andreas Puyskens
Marina Bechtle
Marion Klemm
Ulrike Zedler
Bogdan Silviu Ungureanu
Ute Guhlich-Bornhof
Anne-Britta Koehler
Manuela Stäber
Hans-Joachim Mollenkopf
Robert Hurwitz
Jens Furkert
Gerd Krause
January Weiner
António Jacinto
Ioana Mihai
Maria Leite-de-Moraes
Frank Siebenhaar
Marcus Maurer
Stefan H. E. Kaufmann
机构
[1] Max Planck Institute for Infection Biology,Department of Immunology
[2] Charitéplatz 1,Nuffield Department of Clinical Medicine, Ludwig Institute for Cancer Research
[3] University of Oxford,CEDOC, NOVA Medical School
[4] NOVA University of Lisbon,Research Center of Gastroenterology and Hepatology
[5] Human Genomics Laboratory - University of Medicine and Pharmacy of Craiova,Microarray Core Facility
[6] Leibniz-Forschungsinstitut fuer Molekulare Pharmakologie (FMP),Biochemistry and Protein Purification Core Facility
[7] Robert-Rössle-Strasse 10,Laboratory of Immunoregulation and Immunopathology
[8] University of Medicine and Pharmacy of Craiova,Department of Dermatology and Allergy
[9] Max Planck Institute for Infection Biology,Hagler Institute for Advanced Study
[10] Charitéplatz 1,undefined
[11] Max Planck Institute for Infection Biology. Charitéplatz 1,undefined
[12] INEM (Institut Necker-Enfants Malades),undefined
[13] CNRS UMR8253,undefined
[14] INSERM UMR1151 and Paris Descartes University,undefined
[15] Charité-Universitätsmedizin Berlin,undefined
[16] Texas A&M University,undefined
来源
Scientific Reports | / 9卷
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摘要
As a first host barrier, the skin is constantly exposed to environmental insults that perturb its integrity. Tight regulation of skin homeostasis is largely controlled by the aryl hydrocarbon receptor (AhR). Here, we demonstrate that Henna and its major pigment, the naphthoquinone Lawsone activate AhR, both in vitro and in vivo. In human keratinocytes and epidermis equivalents, Lawsone exposure enhances the production of late epidermal proteins, impacts keratinocyte differentiation and proliferation, and regulates skin inflammation. To determine the potential use of Lawsone for therapeutic application, we harnessed human, murine and zebrafish models. In skin regeneration models, Lawsone interferes with physiological tissue regeneration and inhibits wound healing. Conversely, in a human acute dermatitis model, topical application of a Lawsone-containing cream ameliorates skin irritation. Altogether, our study reveals how a widely used natural plant pigment is sensed by the host receptor AhR, and how the physiopathological context determines beneficial and detrimental outcomes.
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