Association of biomarkers with health-related quality of life and history of stressors in myalgic encephalomyelitis/chronic fatigue syndrome patients

[1] Fukuda K(1994)The chronic fatigue syndrome: a comprehensive approach to its definition and study. International Chronic Fatigue Syndrome Study Group Ann Intern Med 12 953-959

[2] Straus SE(2014)Mitochondrial dysfunctions in myalgic encephalomyelitis/chronic fatigue syndrome explained by activated immune-inflammatory, oxidative and nitrosative stress pathways Metab Brain Dis 29 19-36

[3] Hickie I(2007)Not in the mind of neurasthenic lazybones but in the cell nucleus: patients with chronic fatigue syndrome have increased production of nuclear factor kappa beta Neuro Endocrinol Lett 28 456-462

[4] Sharpe MC(2011)Immunological abnormalities as potential biomarkers in chronic fatigue syndrome/myalgic encephalomyelitis J Transl Med 9 81-355

[5] Dobbins JG(2016)ERK1/2, MEK 1/2, and p38 downstream signaling molecules impaired in CD56(dim)CD16(bright)CD16(dim/-) natural killer cells in chronic fatigue syndrome/myalgic encephalomyelitis patients J Transl Med 14 97-84

[6] Komaroff A(2006)Chronic fatigue syndrome Lancet 367 346-288

[7] Morris G(2012)Chronic fatigue syndrome: acute infection and history of physical activity affect resting levels and response to exercise of plasma oxidant/antioxidant status and heat shock proteins J Intern Med 272 74-310

[8] Maes M(2006)Muscle fatigue and reactive oxygen species J Physiol 576 279-206

[9] Maes M(2005)Chronic fatigue syndrome: assessment of increased oxidative stress and altered muscle excitability in response to incremental exercise J Int Med. 257 299-362

[10] Mihaylova I(2009)Chronic fatigue syndrome combines increased exercise-induced oxidative stress and reduced cytokine and Hsp responses J Intern Med 266 196-2049

[1] Fukuda K(1994)The chronic fatigue syndrome: a comprehensive approach to its definition and study. International Chronic Fatigue Syndrome Study Group Ann Intern Med 12 953-959

[2] Straus SE(2014)Mitochondrial dysfunctions in myalgic encephalomyelitis/chronic fatigue syndrome explained by activated immune-inflammatory, oxidative and nitrosative stress pathways Metab Brain Dis 29 19-36

[3] Hickie I(2007)Not in the mind of neurasthenic lazybones but in the cell nucleus: patients with chronic fatigue syndrome have increased production of nuclear factor kappa beta Neuro Endocrinol Lett 28 456-462

[4] Sharpe MC(2011)Immunological abnormalities as potential biomarkers in chronic fatigue syndrome/myalgic encephalomyelitis J Transl Med 9 81-355

[5] Dobbins JG(2016)ERK1/2, MEK 1/2, and p38 downstream signaling molecules impaired in CD56(dim)CD16(bright)CD16(dim/-) natural killer cells in chronic fatigue syndrome/myalgic encephalomyelitis patients J Transl Med 14 97-84

[6] Komaroff A(2006)Chronic fatigue syndrome Lancet 367 346-288

[7] Morris G(2012)Chronic fatigue syndrome: acute infection and history of physical activity affect resting levels and response to exercise of plasma oxidant/antioxidant status and heat shock proteins J Intern Med 272 74-310

[8] Maes M(2006)Muscle fatigue and reactive oxygen species J Physiol 576 279-206

[9] Maes M(2005)Chronic fatigue syndrome: assessment of increased oxidative stress and altered muscle excitability in response to incremental exercise J Int Med. 257 299-362

[10] Mihaylova I(2009)Chronic fatigue syndrome combines increased exercise-induced oxidative stress and reduced cytokine and Hsp responses J Intern Med 266 196-2049