Network approach identifies Pacer as an autophagy protein involved in ALS pathogenesis

被引:0
|
作者
S. Beltran
M. Nassif
E. Vicencio
J. Arcos
L. Labrador
B. I. Cortes
C. Cortez
C. A. Bergmann
S. Espinoza
M. F. Hernandez
J. M. Matamala
L. Bargsted
S. Matus
D. Rojas-Rivera
M. J. M. Bertrand
D. B. Medinas
C. Hetz
P. A. Manque
U. Woehlbier
机构
[1] Faculty of Science,Center for Integrative Biology
[2] Universidad Mayor,Center for Genomics and Bioinformatics
[3] Faculty of Science,Department of Neurological Sciences
[4] Universidad Mayor,Biomedical Neuroscience Institute, Faculty of Medicine
[5] Faculty of Medicine,Department of Biomedical Molecular Biology
[6] University of Chile,Program of Cellular and Molecular Biology
[7] University of Chile,Department of Immunology and Infectious Diseases
[8] Fundación Ciencia & Vida,Center for the Study of Biological Complexity
[9] Neurounion Biomedical Foundation,undefined
[10] Center for Geroscience,undefined
[11] Brain Health and Metabolism (GERO),undefined
[12] VIB Center for Inflammation Research,undefined
[13] Ghent University,undefined
[14] Buck Institute for Research on Aging,undefined
[15] Institute of Biomedical Sciences,undefined
[16] University of Chile,undefined
[17] Harvard School of Public Health,undefined
[18] Virginia Commonwealth University,undefined
来源
Molecular Neurodegeneration | / 14卷
关键词
ALS; Autophagy; Beclin1; C13orf18, KIAA0226-like; Pacer; Rubicon; Rubicon-like; SOD1; TDP43;
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