Smoking, fat mass and activation of the tumor necrosis factor-α pathway

被引:0
作者
J-M Fernandez-Real
M Broch
J Vendrell
W Ricart
机构
[1] Unitat de Diabetologia,
[2] Endocrinologia i Nutricio,undefined
[3] University Hospital of Girona ‘Dr Josep Trueta’,undefined
[4] Unitat d'Endocrinologia,undefined
[5] Facultat Medicina,undefined
[6] Institut d'Estudis Avançats,undefined
[7] Universitat Rovira i Virgili,undefined
[8] Hospital of Tarragona ‘Joan XXIII’,undefined
来源
International Journal of Obesity | 2003年 / 27卷
关键词
cytokines; obesity; inflammation; insulin resistance; endotoxin;
D O I
暂无
中图分类号
学科分类号
摘要
OBJECTIVE: Obesity may be associated with increased markers of inflammation that could be triggered by metabolic, physical, infectious or environmental processes. As smoking significantly increases cytokine production, we aimed to study how smoking influences the relationship between fat mass and soluble tumor necrosis factor-α (TNF-α) receptors 1 and 2 (sTNFR1 and sTNFR2).
引用
收藏
页码:1552 / 1556
页数:4
相关论文
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