The Fun30 nucleosome remodeller promotes resection of DNA double-strand break ends

被引:0
|
作者
Xuefeng Chen
Dandan Cui
Alma Papusha
Xiaotian Zhang
Chia-Dwo Chu
Jiangwu Tang
Kaifu Chen
Xuewen Pan
Grzegorz Ira
机构
[1] Baylor College of Medicine,Department of Molecular & Human Genetics
[2] One Baylor Plaza,Verna and Marrs McLean Department of Biochemistry and Molecular Biology
[3] Houston,undefined
[4] Texas 77030,undefined
[5] USA,undefined
[6] Baylor College of Medicine,undefined
[7] One Baylor Plaza,undefined
[8] Houston,undefined
[9] Texas 77030,undefined
[10] USA,undefined
[11] Present address: Institute of Plant Protection and Microbiology,undefined
[12] Zhejiang Academy of Agricultural Sciences,undefined
[13] Hangzhou 310021,undefined
[14] China.,undefined
来源
Nature | 2012年 / 489卷
关键词
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中图分类号
学科分类号
摘要
Nucleolytic degradation of 5′ strands at DNA double-strand breaks in yeast is shown to be facilitated by the nucleosome remodeller Fun30, particularly within chromatin bound by the checkpoint adaptor protein known to inhibit resection, Rad9.
引用
收藏
页码:576 / 580
页数:4
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