Haploinsufficiency of interferon regulatory factor 4 strongly protects against autoimmune diabetes in NOD mice

被引:0
|
作者
Satoru Akazawa
Masakazu Kobayashi
Genpei Kuriya
Ichiro Horie
Liping Yu
Hironori Yamasaki
Minoru Okita
Yuji Nagayama
Toshifumi Matsuyama
Masoud Akbari
Katsuyuki Yui
Atsushi Kawakami
Norio Abiru
机构
[1] Nagasaki University Graduate School of Biomedical Sciences,Department of Endocrinology and Metabolism, Unit of Translational Medicine
[2] Nagasaki University,Center for Health and Community Medicine
[3] National Hospital Organization Nagasaki Medical Center,Department of Endocrinology and Metabolism
[4] University of Colorado Health Sciences Center,Barbara Davis Center for Diabetes
[5] Nagasaki University Graduate School of Biomedical Sciences,Department of Locomotive Rehabilitation Science, Unit of Rehabilitation Sciences
[6] Nagasaki University,Department of Molecular Medicine, Atomic Bomb Disease Institute
[7] Nagasaki University Graduate School of Biomedical Sciences,Division of Cytokine Signaling, Department of Molecular Microbiology and Immunology
[8] Nagasaki University Graduate School of Biomedical Sciences,Division of Immunology, Department of Molecular Microbiology and Immunology
来源
Diabetologia | 2015年 / 58卷
关键词
Autoimmunity; Dendritic cells; Interferon regulatory factor 4; NOD mice; T cells; Type 1 diabetes;
D O I
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中图分类号
学科分类号
摘要
引用
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页码:2606 / 2614
页数:8
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