CD36 overexpression: a possible etiopathogenic mechanism of atherosclerosis in patients with prediabetes and diabetes

被引:0
作者
M. D. Lopez-Carmona
M. C. Plaza-Seron
A. Vargas-Candela
F. J. Tinahones
R. Gomez-Huelgas
M. R. Bernal-Lopez
机构
[1] Regional University Hospital of Malaga (Carlos Haya Hospital),Internal Medicine Department, Biomedical Institute of Malaga (IBIMA)
[2] Regional University Hospital of Malaga (Carlos Haya Hospital),Research Laboratory
[3] Regional University Hospital of Malaga (Virgen de la Victoria Hospital),Allergy Unit, Biomedical Institute of Malaga (IBIMA)
[4] CIBERFisiopatología de la Obesidad y Nutrición,Endocrinology and Nutrition Department, Biomedical Institute of Malaga (IBIMA)
[5] Instituto de Salud Carlos III,undefined
来源
Diabetology & Metabolic Syndrome | / 9卷
关键词
Atherosclerosis; Type 2 diabetes; CD36 receptor; Human clinical; Monocytes;
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  • [1] Cooper R(2000)Trends and disparities in coronary heart disease, stroke, and other cardiovascular diseases in the United States: findings of the national conference on cardiovascular disease prevention Circulation 102 3137-3147
  • [2] Cutler J(2013)Trends in predicted 10-year risk of coronary heart disease and cardiovascular disease among US adults from 1999 to 2010 J Am Coll Cardiol 61 2249-2252
  • [3] Desvigne-Nickens P(2010)Standards of medical care in diabetes–2010 Diabetes Care. 33 S4-S10
  • [4] Fortmann SP(2012)Insulin promotes macrophage foam cell formation: potential implications in diabetes-related atherosclerosis Lab Invest 92 1171-1180
  • [5] Friedman L(1999)Glucose tolerance and mortality: comparison of WHO and American Diabetes Association diagnostic criteria. Diabetes epidemiology: Collaborative analysis Of Diagnostic criteria in Europe Lancet. 354 617-621
  • [6] Havlik R(1991)Distribution patterns of apolipoproteins A1, A2, and B in the wall of atherosclerotic vessels Virchows Arch A Pathol Anat Histopathol. 419 79-88
  • [7] Ford ES(1989)Initiation of atherosclerotic lesions in cholesterol-fed rabbits. II. Selective retention of LDL vs. selective increases in LDL permeability in susceptible sites of arteries Arteriosclerosis. 9 908-918
  • [8] Park YM(1989)Initiation of atherosclerotic lesions in cholesterol-fed rabbits. I. Focal increases in arterial LDL concentration precede development of fatty streak lesions Arteriosclerosis. 9 895-907
  • [9] Kashyap SR(1990)Expression of the acetyl low density lipoprotein receptor by rabbit fibroblasts and smooth muscle cells. Up-regulation by phorbol esters J Biol Chem 265 12722-12727
  • [10] Major JA(1977)The low-density lipoprotein pathway and its relation to atherosclerosis Annu Rev Biochem 46 897-930