Post-CNS-inflammation expression of CXCL12 promotes the endogenous myelin/neuronal repair capacity following spontaneous recovery from multiple sclerosis-like disease

被引:0
|
作者
Rina Zilkha-Falb
Nathali Kaushansky
Naoto Kawakami
Avraham Ben-Nun
机构
[1] The Weizmann Institute of Science,Department of Immunology
[2] Ludwig-Maximilians-University,Institute of Clinical Neuroimmunology
[3] Sheba Medical Center,Present address: Multiple Sclerosis Center, Neurogenomics Laboratory
关键词
Multiple sclerosis; Experimental autoimmune encephalomyelitis (EAE); CXCL12; Neuronal progenitor cells; Oligodendrocyte precursor cells; Myelin and neuronal repair;
D O I
暂无
中图分类号
学科分类号
摘要
引用
收藏
相关论文
共 1 条