Multi-omics analyses of MEN1 missense mutations identify disruption of menin–MLL and menin–JunD interactions as critical requirements for molecular pathogenicity

被引：0

作者：

Koen M. A. Dreijerink

Ezgi Ozyerli-Goknar

Stefanie Koidl

Ewoud J. van der Lelij

Priscilla van den Heuvel

Jeffrey J. Kooijman

Martin L. Biniossek

Kees W. Rodenburg

Sheikh Nizamuddin

H. T. Marc Timmers

机构：

[1] Amsterdam UMC,Department of Endocrinology

[2] German Cancer Research Center (DKFZ),German Cancer Consortium (DKTK) Partner Site Freiburg

[3] Medical Center -University of Freiburg,Department of Urology

[4] Avans University of Applied Sciences,School of Life Sciences, and Research Group of Technologies of Analyses in Life Sciences (ATLS)

[5] University of Freiburg,Institute for Molecular Medicine and Cell Research

[6] Oncolines B.V.,undefined

来源：

Epigenetics & Chromatin | / 15卷

关键词：

Tumor suppressor; MEN1 syndrome; Histone H3K4 methylation; AP-1 transcription factors; ATF transcription factors; Quantitative mass spectrometry; CUT&RUN genome profiling;

D O I：

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摘要：

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