Long-term exercise results in morphological and biomechanical changes in coronary resistance arterioles in male and female rats

[1] Radovits T(2013)Rat model of exercise-induced cardiac hypertrophy: hemodynamic characterization using left ventricular pressure-volume analysis Am J Physiol Heart Circ Physiol 305 H124-H134

[2] Olah A(2015)Strain and strain rate by speckle-tracking echocardiography correlate with pressure-volume loop-derived contractility indices in a rat model of athlete's heart Am J Physiol Heart Circ Physiol 308 H743-H748

[3] Lux A(2016)Physiological and pathological left ventricular hypertrophy of comparable degree is associated with characteristic differences of in vivo hemodynamics Am J Physiol Heart Circ Physiol 310 H587-H597

[4] Nemeth BT(2010)Molecular distinction between physiological and pathological cardiac hypertrophy: experimental findings and therapeutic strategies Pharmacol Ther 128 191-227

[5] Hidi L(2012)Physiological cardiac remodelling in response to endurance exercise training: cellular and molecular mechanisms Heart 98 5-10

[6] Birtalan E(2003)Phosphoinositide 3-kinase (p110alpha) plays a critical role for the induction of physiological, but not pathological, cardiac hypertrophy Proc Natl Acad Sci U S A 100 12355-12360

[7] Kovacs A(2019)Characterization of the dynamic changes in left ventricular morphology and function induced by exercise training and detraining Int J Cardiol 277 178-185

[8] Olah A(2017)Complete reversion of cardiac functional adaptation induced by exercise training Med Sci Sports Exerc 49 420-429

[9] Lux A(2018)Differential cardiac hypertrophy and signaling pathways in pressure versus volume overload Am J Physiol Heart Circ Physiol 314 H552-Hh62

[10] Matyas C(2017)Myocardial fibrosis and cardiac decompensation in aortic stenosis JACC Cardiovasc Imaging 10 1320-1333

[1] Radovits T(2013)Rat model of exercise-induced cardiac hypertrophy: hemodynamic characterization using left ventricular pressure-volume analysis Am J Physiol Heart Circ Physiol 305 H124-H134

[2] Olah A(2015)Strain and strain rate by speckle-tracking echocardiography correlate with pressure-volume loop-derived contractility indices in a rat model of athlete's heart Am J Physiol Heart Circ Physiol 308 H743-H748

[3] Lux A(2016)Physiological and pathological left ventricular hypertrophy of comparable degree is associated with characteristic differences of in vivo hemodynamics Am J Physiol Heart Circ Physiol 310 H587-H597

[4] Nemeth BT(2010)Molecular distinction between physiological and pathological cardiac hypertrophy: experimental findings and therapeutic strategies Pharmacol Ther 128 191-227

[5] Hidi L(2012)Physiological cardiac remodelling in response to endurance exercise training: cellular and molecular mechanisms Heart 98 5-10

[6] Birtalan E(2003)Phosphoinositide 3-kinase (p110alpha) plays a critical role for the induction of physiological, but not pathological, cardiac hypertrophy Proc Natl Acad Sci U S A 100 12355-12360

[7] Kovacs A(2019)Characterization of the dynamic changes in left ventricular morphology and function induced by exercise training and detraining Int J Cardiol 277 178-185

[8] Olah A(2017)Complete reversion of cardiac functional adaptation induced by exercise training Med Sci Sports Exerc 49 420-429

[9] Lux A(2018)Differential cardiac hypertrophy and signaling pathways in pressure versus volume overload Am J Physiol Heart Circ Physiol 314 H552-Hh62

[10] Matyas C(2017)Myocardial fibrosis and cardiac decompensation in aortic stenosis JACC Cardiovasc Imaging 10 1320-1333