The transcriptional responsiveness of LKB1 to STAT-mediated signaling is differentially modulated by prolactin in human breast cancer cells

被引：0
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作者：
Katja Linher-Melville
Gurmit Singh
机构：
[1] McMaster University,Department of Pathology and Molecular Medicine
来源：
BMC Cancer | / 14卷
关键词：
Breast cancer; STAT3; STAT5; LKB1; Prolactin; Interleukin 6; Promoter; Transcriptional regulation;
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