Clinical case report: Falciparum malaria with hemophagocytic syndrome

被引:15
作者
Ohnishi, Kenji
Mitsui, Keiko
Komiya, Nobuhiro
Iwasaki, Noriaki
Akashi, Akiko
Hamabe, Yuichi
机构
[1] Tokyo Metropolitan Bukutoh Gen Hosp, Dept Infect Dis, Sumida City, Tokyo 1308575, Japan
[2] Tokyo Metropolitan Bukutoh Gen Hosp, Teritary Emergency Med Ctr, Sumida City, Tokyo 1308575, Japan
关键词
D O I
10.4269/ajtmh.2007.76.1016
中图分类号
R1 [预防医学、卫生学];
学科分类号
1004 ; 120402 ;
摘要
A 30-year-old Japanese woman with falciparum malaria was hospitalized because of fever and renal failure, and prolonged anemia was identified despite the eradication of malaria parasites through anti-malaria therapy. Bone marrow aspiration revealed the presence of macrophages with hemophagocytosis, and serum interleukin (IL)-18 and tumor necrosis factor (TNF)-alpha levels were high in the anemic phase. Hemophagocytosis was confirmed for at least 3 weeks, and prolonged hemophagocytic syndrome is thought to be one of the causes of prolonged anemia in patients with falciparum malaria.
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收藏
页码:1016 / 1018
页数:3
相关论文
共 17 条
[1]  
Abdelkefi Abderrahman, 2004, Hematol J, V5, P449, DOI 10.1038/sj.thj.6200531
[2]  
Anwar Masood, 1995, JPMA (Journal of the Pakistan Medical Association), V45, P302
[3]   Haemophagocytic syndrome associated with Plasmodium vivax infection [J].
Aouba, A ;
Noguera, ME ;
Clauvel, JP ;
Quint, L .
BRITISH JOURNAL OF HAEMATOLOGY, 2000, 108 (04) :832-833
[4]   TUMOR NECROSIS FACTOR MAY CONTRIBUTE TO THE ANEMIA OF MALARIA BY CAUSING DYSERYTHROPOIESIS AND ERYTHROPHAGOCYTOSIS [J].
CLARK, IA ;
CHAUDHRI, G .
BRITISH JOURNAL OF HAEMATOLOGY, 1988, 70 (01) :99-103
[5]  
Fujiki Rei, 2003, Kekkaku, V78, P443
[6]  
Goto S, 2001, AM J KIDNEY DIS, V38, part. no.
[7]   FALCIPARUM-MALARIA WITH BONE-MARROW ABNORMALITIES RESEMBLING MALIGNANT HISTIOCYTOSIS [J].
HAMADA, A ;
WATANABE, N ;
TANAKA, H ;
KOBAYASHI, A .
TRANSACTIONS OF THE ROYAL SOCIETY OF TROPICAL MEDICINE AND HYGIENE, 1989, 83 (03) :331-331
[8]  
KOIZUMI K, 1996, JPN J CLIN HEMATOL, V37, P40
[9]   Upregulation of tumor necrosis factor-alpha gene by Epstein-Barr virus and activation of macrophages in Epstein-Barr virus-infected T cells in the pathogenesis of hemophagocytic syndrome [J].
Lay, JD ;
Tsao, CJ ;
Chen, JY ;
Kadin, ME ;
Su, IJ .
JOURNAL OF CLINICAL INVESTIGATION, 1997, 100 (08) :1969-1979
[10]  
Ohno T, 1996, INT J HEMATOL, V64, P263