Neuronal degeneration and mitochondrial dysfunction

[1] Frontal lobe dysfunction in progressive supranuclear palsy: Evidence for oxidative stress and mitochondrial impairment [J]. JOURNAL OF NEUROCHEMISTRY, 2000, 74 (02) : 878 - 881

[2] SEQUENCE AND ORGANIZATION OF THE HUMAN MITOCHONDRIAL GENOME [J]. NATURE, 1981, 290 (5806) : 457 - 465

[3] CREB activation induced by mitochondrial dysfunction is a new signaling pathway that impairs cell proliferation [J]. EMBO JOURNAL, 2002, 21 (1-2) : 53 - 63

[4] AGE-DEPENDENT STRIATAL EXCITOTOXIC LESIONS PRODUCED BY THE ENDOGENOUS MITOCHONDRIAL INHIBITOR MALONATE [J]. JOURNAL OF NEUROCHEMISTRY, 1993, 61 (03) : 1147 - 1150

[5] Mitochondrial involvement in Alzheimer's disease [J]. BIOCHIMICA ET BIOPHYSICA ACTA-BIOENERGETICS, 1999, 1410 (02): : 171 - 182

[6] Borthwick GM, 1999, ANN NEUROL, V46, P787, DOI 10.1002/1531-8249(199911)46:5<787::AID-ANA17>3.0.CO

[7] 2-8

[8] Browne SE, 1998, J NEUROCHEM, V71, P281

[9] Human skeletal muscle cytosols are refractory to cytochrome c-dependent activation of type-II caspases and lack APAF-1 [J]. CELL DEATH AND DIFFERENTIATION, 1999, 6 (03) : 256 - 261

[10] Altered metabolism of the amyloid β precursor protein is associated with mitochondrial dysfunction in Down's syndrome [J]. NEURON, 2002, 33 (05) : 677 - 688

[1] Frontal lobe dysfunction in progressive supranuclear palsy: Evidence for oxidative stress and mitochondrial impairment [J]. JOURNAL OF NEUROCHEMISTRY, 2000, 74 (02) : 878 - 881

[2] SEQUENCE AND ORGANIZATION OF THE HUMAN MITOCHONDRIAL GENOME [J]. NATURE, 1981, 290 (5806) : 457 - 465

[3] CREB activation induced by mitochondrial dysfunction is a new signaling pathway that impairs cell proliferation [J]. EMBO JOURNAL, 2002, 21 (1-2) : 53 - 63

[4] AGE-DEPENDENT STRIATAL EXCITOTOXIC LESIONS PRODUCED BY THE ENDOGENOUS MITOCHONDRIAL INHIBITOR MALONATE [J]. JOURNAL OF NEUROCHEMISTRY, 1993, 61 (03) : 1147 - 1150

[5] Mitochondrial involvement in Alzheimer's disease [J]. BIOCHIMICA ET BIOPHYSICA ACTA-BIOENERGETICS, 1999, 1410 (02): : 171 - 182

[6] Borthwick GM, 1999, ANN NEUROL, V46, P787, DOI 10.1002/1531-8249(199911)46:5<787::AID-ANA17>3.0.CO

[7] 2-8

[8] Browne SE, 1998, J NEUROCHEM, V71, P281

[9] Human skeletal muscle cytosols are refractory to cytochrome c-dependent activation of type-II caspases and lack APAF-1 [J]. CELL DEATH AND DIFFERENTIATION, 1999, 6 (03) : 256 - 261

[10] Altered metabolism of the amyloid β precursor protein is associated with mitochondrial dysfunction in Down's syndrome [J]. NEURON, 2002, 33 (05) : 677 - 688