Rapid induction of myeloid leukemia by co-expressing BCR-ABL and AML1/MDS1/EVI1 fusion genes.

被引：0

作者：

Cuenco, GM ^{[1
]}

Joyce, J ^{[1
]}

Ren, RB ^{[1
]}

机构：

[1] Brandeis Univ, Dept Biol, Waltham, MA 02254 USA

来源：

BLOOD | 2000年 / 96卷 / 11期

关键词：

D O I：

暂无

中图分类号：

R5 [内科学];

学科分类号：

1002 ; 100201 ;

摘要：

1975

引用

页码：459A / 459A

页数：1

共 50 条

[1] Cooperation of BCR-ABL and AML1/MDS1/EVI1 in blocking myeloid differentiation and rapid induction of an acute myelogenous leukemia
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[2] Cooperation of BCR-ABL and AML1/MDS1/EVI1 in blocking myeloid differentiation and rapid induction of an acute myelogenous leukemia
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[3] Both AML1 and EVI1 oncogenic components are required for the cooperation of AML1/MDS1/EVI1 with BCR/ABL in the induction of acute myelogenous leukemia in mice
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[10] P/CAF and GCN5 acetylate the AML1/MDS1/EVI1 fusion oncoprotein
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