Disruption of Epac1 gene preserves cardiac function against pressure overload and chronic catecholamine stress

被引:0
|
作者
Jin, Meihua [1 ]
Okumura, Satoshi [1 ]
Cai, Wenqian [1 ]
Hidaka, Yuko [1 ]
Kurotani, Reiko [1 ]
Yokoyama, Utako [1 ]
Sato, Motohiko [1 ]
Ishikawa, Yoshihiro [1 ]
机构
[1] Yokohama City Univ, Grad Sch Med, Cardiovasc Res Inst, Yokohama, Kanagawa, Japan
来源
JOURNAL OF PHYSIOLOGICAL SCIENCES | 2010年 / 60卷
关键词
D O I
暂无
中图分类号
Q4 [生理学];
学科分类号
071003 ;
摘要
引用
收藏
页码:S163 / S163
页数:1
相关论文
共 50 条
  • [21] Novel mechanisms for caspase inhibition protecting cardiac function with chronic pressure overload
    Park, Misun
    Vatner, Stephen F.
    Yan, Lin
    Gao, Shumin
    Yoon, Seunghun
    Lee, Grace Jung Ah
    Xie, Lai-Hua
    Kitsis, Richard N.
    Vatner, Dorothy E.
    BASIC RESEARCH IN CARDIOLOGY, 2013, 108 (01)
  • [22] Increased angiogenesis as a mechanism for the preserved cardiac function in rats with chronic pressure overload
    Gao, Shumin
    Park, Misun
    Tian, Yimin
    Yan, Lin
    Lin, Lin
    Zhao, Xin
    Gelpi, Ricardo
    Vatner, Dorothy E.
    Vatner, Stephen F.
    FASEB JOURNAL, 2012, 26
  • [23] Chronic Pressure Overload Impacts Cardiac Endothelial Cell and Macrophage Gene Expression
    Lother, Achim
    Deng, Lisa
    Hilgendorf, Ingo
    Kehl, Theresa
    Bode, Christoph
    Hein, Lutz
    HYPERTENSION, 2018, 72
  • [24] Caspase Inhibition Prevents Hypertrophy and Improves Cardiac Function with Chronic Pressure Overload
    Park, Misun
    Lee, Jung Ah
    Gao, Shumin
    Yoon, SeongHun
    Xie, Lai-Hua
    Charles, Ludny
    Leibovich, Samuel J.
    Kitsis, Richard N.
    Vatner, Stephen F.
    Vatner, Dorothy E.
    CIRCULATION, 2011, 124 (21)
  • [25] Maintaining PGC-1α expression following pressure overload-induced cardiac hypertrophy preserves angiogenesis but not contractile or mitochondrial function
    Pereira, Renata O.
    Wende, Adam R.
    Crum, Ashley
    Hunter, Douglas
    Olsen, Curtis D.
    Rawlings, Tenley
    Riehle, Christian
    Ward, Walter F.
    Abel, E. Dale
    FASEB JOURNAL, 2014, 28 (08): : 3691 - 3702
  • [26] Extracellular signal-regulated kinase (ERK) activation preserves cardiac function in pressure overload induced hypertrophy
    Mutlak, Michael
    Schlesinger-Laufer, Michal
    Haas, Tali
    Shofti, Rona
    Ballan, Nimer
    Lewis, Yair E.
    Zuler, Mor
    Zohar, Yaniv
    Caspi, Lilac H.
    Kehat, Izhak
    INTERNATIONAL JOURNAL OF CARDIOLOGY, 2018, 270 : 204 - 213
  • [27] Still stressed out but doing fine - Normalization of wall stress is superfluous to maintaining cardiac function in chronic pressure overload
    Sano, M
    Schneider, MD
    CIRCULATION, 2002, 105 (01) : 8 - 10
  • [28] Melusin cardiac overexpression protects against dilated cardiomyopathy due to chronic pressure overload
    Notte, A
    De Acetis, M
    Selvetella, G
    Brancaccio, M
    Russo, G
    Tarone, G
    Lembo, G
    JOURNAL OF HYPERTENSION, 2004, 22 : S172 - S172
  • [29] Taxifolin protects against cardiac hypertrophy and fibrosis during biomechanical stress of pressure overload
    Guo, Haipeng
    Zhang, Xin
    Cui, Yuqian
    Zhou, Heng
    Xu, Dachun
    Shan, Tichao
    Zhang, Fan
    Guo, Yuan
    Chen, Yuguo
    Wu, Dawei
    TOXICOLOGY AND APPLIED PHARMACOLOGY, 2015, 287 (02) : 168 - 177
  • [30] Prevention of pressure overload hypertrophy with preserved function by β1/β2-ADRENocepTOR disruption
    Wang, K
    Gao, XM
    Moore, XL
    Gibbs, ME
    Dart, AM
    Du, XJ
    Kiriazis, H
    JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY, 2005, 39 (03) : 574 - 575