Loss of RAF kinase inhibitor protein is a somatic event in the pathogenesis of therapy-related acute myeloid leukemias with C-RAF germline mutations

被引：0

作者：

Haller, M. ^{[1
]}

Zebisch, A. ^{[2
]}

Hiden, K. ^{[3
]}

Hoefler, G. ^{[4
]}

Sill, H. ^{[3
]}

Troppmair, J. ^{[1
]}

机构：

[1] Innsbruck Med Univ, Daniel Swarovski Res Lab, Innsbruck, Austria

[2] Beatson Inst Canc Res, Glasgow G61 1BD, Lanark, Scotland

[3] Med Univ Graz, Div Hematol, Graz, Austria

[4] Med Univ Graz, Inst Pathol, Graz, Austria

来源：

FEBS JOURNAL | 2010年 / 277卷

关键词：

D O I：

暂无

中图分类号：

Q5 [生物化学]; Q7 [分子生物学];

学科分类号：

071010 ; 081704 ;

摘要：

引用

页码：127 / 127

页数：1

共 21 条

[1] Loss of RAF kinase inhibitor protein is a somatic event in the pathogenesis of therapy-related acute myeloid leukemias with C-RAF germline mutations
A Zebisch
M Haller
K Hiden
T Goebel
G Hoefler
J Troppmair
H Sill
Leukemia, 2009, 23 : 1049 - 1053
[2] Loss of RAF kinase inhibitor protein is a somatic event in the pathogenesis of therapy-related acute myeloid leukemias with C-RAF germline mutations
Zebisch, A.
Haller, M.
Hiden, K.
Goebel, T.
Hoefler, G.
Troppmair, J.
Sill, H.
LEUKEMIA, 2009, 23 (06) : 1049 - 1053
[3] Two transforming C-RAF germ-line mutations identified in patients with therapy-related acute myeloid leukemia
Zebisch, A
Staber, PB
Delavar, A
Bodner, C
Hiden, K
Fischereder, K
Janakiraman, M
Linkesch, W
Auner, HW
Emberger, W
Windpassinger, C
Schimek, MG
Hoefler, G
Troppmair, J
Sill, H
CANCER RESEARCH, 2006, 66 (07) : 3401 - 3408
[4] Two novel activating germline mutations of the C-RAF proto-oncogene predisposing to solid tbmors and therapy-related acute myeloid leukemia.
Zebisch, A
Staber, PB
Fischereder, K
Bodner, C
Hiden, K
Linkesch, W
Auner, HW
Emberger, W
Windpassinger, C
Schimek, MG
Hoefler, G
Troppmair, J
Sill, H
BLOOD, 2004, 104 (11) : 920A - 920A
[5] Frequent loss of RAF kinase inhibitor protein expression in acute myeloid leukemia
Zebisch, A.
Woelfler, A.
Fried, I.
Wolf, O.
Lind, K.
Bodner, C.
Haller, M.
Drasche, A.
Pirkebner, D.
Matallanas, D.
Rath, O.
Blyth, K.
Delwel, R.
Taskesen, E.
Quehenberger, F.
Kolch, W.
Troppmair, J.
Sill, H.
LEUKEMIA, 2012, 26 (08) : 1842 - 1849
[6] Frequent loss of RAF kinase inhibitor protein expression in acute myeloid leukemia
A Zebisch
A Wölfler
I Fried
O Wolf
K Lind
C Bodner
M Haller
A Drasche
D Pirkebner
D Matallanas
O Rath
K Blyth
R Delwel
E Taskesen
F Quehenberger
W Kolch
J Troppmair
H Sill
Leukemia, 2012, 26 : 1842 - 1849
[7] Loss of RAF Kinase Inhibitor Protein Is a Frequent Event In Acute Myeloid Leukemia with a Monocytic Phenotype and Cooperates with Mutant RAS In Malignant Transformation
Zebisch, Armin
Woelfler, Albert
Hiden, Karin
Bodner, Claudia
Quehenberger, Franz
Drasche, Astrid
Pirkebner, Daniela
Haller, Martina
Rath, Oliver
Blyth, Karen
Taskesen, Erdogan
Kolch, Walter
Troppmair, Jakob
Sill, Heinz
BLOOD, 2010, 116 (21) : 1702 - 1702
[8] The RAF kinase inhibitor protein acts as a novel tumor suppressor in acute myeloid leukemia
Zebisch, A.
Woelfler, A.
Hiden, K.
Bodner, C.
Quehenberger, F.
Drasche, A.
Pirkebner, D.
Haller, M.
Matallanas, D.
Fried, I.
Rath, O.
Blyth, K.
Taskesen, E.
Kolch, W.
Troppmair, J.
H., Sill
ONKOLOGIE, 2011, 34 : 209 - 209
[9] Final analysis of a randomized phase I study of the C-RAF kinase inhibitor BAY 43-9006 in patients with acute myeloid leukemia
Crump, Michael
Leber, Brian
Buckstein, Rena
Kassis, Jeanine
Headley, David
Minden, Mark
Petrenciuc, Oana
Maclntosh, Lynn
Seymour, Leslet
Eisenhauer, Elizabeth
ANNALS OF ONCOLOGY, 2004, 15 : 154 - 154
[10] Loss of RAF kinase inhibitor protein is involved in myelomonocytic differentiation an aggravates RAS-driven myeloid leukemogenesis
Caraffini, Veronica
Geiger, Olivia
Rosenberger, Angelika
Hatzl, Stefan
Perfler, Bianca
Berg, Johannes L.
Lim, Clarice
Strobl, Herbert
Kashofer, Karl
Schauer, Silvia
Beham-Schmid, Christine
Hoefler, Gerald
Geissler, Klaus
Quehenberger, Franz
Kolch, Walter
Athineos, Dimitris
Blyth, Karen
Woelfler, Albert
Sill, Heinz
Zebisch, Armin
HAEMATOLOGICA, 2020, 105 (02) : 375 - 386

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