To Die or Not to Die, That's the Question - And the Answer May Depend on Netrin-1

被引：3

作者：

Schoen, Michael P. ^{[1
]}

Schoen, Margarete ^{[2
]}

机构：

[1] Univ Gottingen, Dept Dermatol & Venereol, D-37075 Gottingen, Germany

[2] Univ Gottingen, Dept Mol & Cellular Immunol, D-37075 Gottingen, Germany

来源：

JNCI-JOURNAL OF THE NATIONAL CANCER INSTITUTE | 2009年 / 101卷 / 04期

关键词：

TUMOR-CELL; DCC GENE; APOPTOSIS; RECEPTORS; MECHANISM; EXPRESSION; GUIDANCE; DEATH;

D O I：

10.1093/jnci/djn511

中图分类号：

R73 [肿瘤学];

学科分类号：

100214 ;

摘要：

引用

页数：3

共 26 条

[1] Netrin-1 and its receptors in tumorigenesis [J].

Arakawa, H .

NATURE REVIEWS CANCER, 2004, 4 (12) :978-987

[2] To kill a tumor cell: the potential of proapoptotic receptor agonists [J].

Ashkenazi, Avi ;

Herbst, Roy S. .

JOURNAL OF CLINICAL INVESTIGATION, 2008, 118 (06) :1979-1990

[3] The RET proto-oncogene induces apoptosis: a novel mechanism for Hirschsprung disease [J].

Bordeaux, MC ;

Forcet, C ;

Granger, L ;

Corset, V ;

Bidaud, C ;

Billaud, M ;

Bredesen, DE ;

Edery, P ;

Mehlen, P .

EMBO JOURNAL, 2000, 19 (15) :4056-4063

[4]

Chalah A, 2008, ADV EXP MED BIOL, V615, P25, DOI 10.1007/978-1-4020-6554-5_3

[5]

Del Poeta G, 2008, CURR CANCER DRUG TAR, V8, P207

[6] Interference With Netrin-1 and Tumor Cell Death in Non-Small Cell Lung Cancer [J].

Delloye-Bourgeois, Celine ;

Brambilla, Elisabeth ;

Coissieux, Marie-May ;

Guenebeaud, Celine ;

Pedeux, Remy ;

Firlej, Virginie ;

Cabon, Florence ;

Brambilla, Christian ;

Mehlen, Patrick ;

Bernet, Agnes .

JNCI-JOURNAL OF THE NATIONAL CANCER INSTITUTE, 2009, 101 (04) :237-247

[7] Proliferation, cell cycle and apoptosis in cancer [J].

Evan, GI ;

Vousden, KH .

NATURE, 2001, 411 (6835) :342-348

[8] IDENTIFICATION OF A CHROMOSOME-18Q GENE THAT IS ALTERED IN COLORECTAL CANCERS [J].

FEARON, ER ;

CHO, KR ;

NIGRO, JM ;

KERN, SE ;

SIMONS, JW ;

RUPPERT, JM ;

HAMILTON, SR ;

PREISINGER, AC ;

THOMAS, G ;

KINZLER, KW ;

VOGELSTEIN, B .

SCIENCE, 1990, 247 (4938) :49-56

[9] Netrin-1-mediated axon outgrowth requires deleted in colorectal cancer-dependent MAPK activation [J].

Forcet, C ;

Stein, E ;

Pays, L ;

Corset, R ;

Llambi, F ;

Tessler-Lavigne, M ;

Mehlen, P .

NATURE, 2002, 417 (6887) :443-447

[10] The dependence receptor DCC (deleted in colorectal cancer) defines an alternative mechanism for caspase activation [J].

Forcet, C ;

Ye, X ;

Granger, L ;

Corset, V ;

Shin, H ;

Bredesen, DE ;

Mehlen, P .

PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 2001, 98 (06) :3416-3421

← 1 2 3 →