NOD2 AND ATG16 L1 ARE ASSOCIATED WITH ANTI-TNF TROUGH LEVELS IN A COHORT OF PATIENTS WITH CROHN'S DISEASE

被引：0
|
作者：
Almenara, S. [1 ]
Egoavil, C. M. [1 ]
Londono, A. C. [1 ]
Gutierrez, A. [2 ,3 ]
Sempere, L. [2 ,3 ]
Zapater, P. [1 ,2 ,3 ]
Cameo, J., I [3 ]
Gonzalez-Navajas, J. [2 ]
Frances, R. [2 ]
机构：
[1] Hosp Gen Univ Alicante, Serv Farmacol Clin, Alicante, Spain
[2] Inst Salud Carlos III, CIBERehd, Madrid, Spain
[3] Hosp Gen Univ Alicante, Serv Med Digest, Alicante, Spain
来源：
BASIC & CLINICAL PHARMACOLOGY & TOXICOLOGY | 2018年 / 123卷
关键词：
anti-TNF levels; Crohn's disease; pharmacogenetics;
D O I：
暂无
中图分类号：
R9 [药学];
学科分类号：
1007 ;
摘要：
P158
引用
收藏
页码：94 / 95
页数：2
相关论文
共 50 条
  • [1] ATG16L1 and NOD2 polymorphisms enhance phagocytosis in monocytes of Crohn's disease patients
    Wolfkamp, Simone C. S.
    Verseyden, Caroline
    Vogels, Esther W. M.
    Meisner, Sander
    Boonstra, Kirsten
    Peters, Charlotte P.
    Stokkers, Pieter C. F.
    te Velde, Anje A.
    WORLD JOURNAL OF GASTROENTEROLOGY, 2014, 20 (10) : 2664 - 2672
  • [2] ATG16L1 and NOD2 polymorphisms enhance phagocytosis in monocytes of Crohn's disease patients
    Simone CS Wolfkamp
    Caroline Verseyden
    Esther WM Vogels
    Sander Meisner
    Kirsten Boonstra
    Charlotte P Peters
    Pieter CF Stokkers
    Anje A te Velde
    World Journal of Gastroenterology, 2014, (10) : 2664 - 2672
  • [3] NOD2 and ATG16L1 polymorphisms affect monocyte responses in Crohn's disease
    Dylan M Glubb
    Richard B Gearry
    Murray L Barclay
    Rebecca L Roberts
    John Pearson
    Jacqui I Keenan
    Judy McKenzie
    Robert W Bentley
    World Journal of Gastroenterology, 2011, 17 (23) : 2829 - 2837
  • [4] NOD2 and ATG16L1 polymorphisms affect monocyte responses in Crohn's disease
    Glubb, Dylan M.
    Gearry, Richard B.
    Barclay, Murray L.
    Roberts, Rebecca L.
    Pearson, John
    Keenan, Jacqui I.
    McKenzie, Judy
    Bentley, Robert W.
    WORLD JOURNAL OF GASTROENTEROLOGY, 2011, 17 (23) : 2829 - 2837
  • [5] Crohn's disease associated ATG16L1 polymorphism modulates cytokine production after NOD2 engagement
    Plantinga, Theo S.
    Crisan, Tania
    Oosting, Marije
    van de Veerdonk, Frank L.
    Kullberg, Bart-Jan
    de Jong, Dirk J.
    Philpott, Dana J.
    Girardin, Stephen E.
    Joosten, Leo A. B.
    Netea, Mihai G.
    CYTOKINE, 2010, 52 (1-2) : 55 - 56
  • [6] ATG16L1 Genotype Is Associated With Response to Anti-TNF
    Wildenberg, Manon E.
    Levin, Alon D.
    Brandse, Johannan F.
    de Bruyn, Jessica R.
    D'Haens, Geert R.
    van den Brink, Gijs R.
    GASTROENTEROLOGY, 2014, 146 (05) : S876 - S877
  • [7] Role of ATG16L,NOD2 and IL23R in Crohn's disease pathogenesis
    Saleh A Naser
    Melissa Arce
    Anam Khaja
    Marlene Fernandez
    Najih Naser
    Sammer Elwasila
    Saisathya Thanigachalam
    World Journal of Gastroenterology, 2012, 18 (05) : 412 - 424
  • [8] Role of Mutations in NOD2/CARD15, ATG16L1, and IRGM in the Pathogenesis of Crohn's Disease
    Maev, Igor' V.
    Andreev, Dmitry N.
    INTERNATIONAL JOURNAL OF BIOMEDICINE, 2014, 4 (01) : 7 - 10
  • [9] Role of ATG16L, NOD2 and IL23R in Crohn's disease pathogenesis
    Naser, Saleh A.
    Arce, Melissa
    Khaja, Anam
    Fernandez, Marlene
    Naser, Najih
    Elwasila, Sammer
    Thanigachalam, Saisathya
    WORLD JOURNAL OF GASTROENTEROLOGY, 2012, 18 (05) : 412 - 424
  • [10] ATG16L1 and NOD2 Interact in an Autophagy-Dependent Anti-Bacterial Pathway Implicated in Crohn's Disease Pathogenesis
    Homer, Craig R.
    Richmond, Amy L.
    Rebert, Nancy A.
    Achkar, Jean-Paul
    McDonald, Christine
    GASTROENTEROLOGY, 2010, 138 (05) : S681 - S681
12345