Responses to eating: lipoproteins, lipolytic products and atherosclerosis

被引：36

作者：

Goldberg, IJ ^{[1
]}

Kako, Y ^{[1
]}

Lutz, EP ^{[1
]}

机构：

[1] Columbia Univ Coll Phys & Surg, Dept Med, Div Prevent Med & Nutr, New York, NY 10032 USA

来源：

CURRENT OPINION IN LIPIDOLOGY | 2000年 / 11卷 / 03期

关键词：

D O I：

10.1097/00041433-200006000-00003

中图分类号：

Q5 [生物化学]; Q7 [分子生物学];

学科分类号：

071010 ; 081704 ;

摘要：

Several lines of clinical and experimental data suggest that postprandial lipemia is an independent risk factor for atherosclerosis. There are a number of reasons why processes that occur in the period immediately after eating could be deleterious to arteries. By understanding the links between postprandial lipemia and the accumulation of lipid within vessels, a more global understanding of how lipoproteins cause disease may be forthcoming. In this article recent information on the control of postprandial lipemia and the biological effects of chylomicron remnants and lipolysis products will be reviewed. Because this topic is broad, we will focus on the roles played by lipoprotein lipase and proteoglycans in this process. Curr Opin Lipidol 11:235-241, (C) 2000 Lippincott Williams & Wilkins.

引用

页码：235 / 241

页数：7

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