Irbesartan inhibits advanced glycation end product-induced increase in asymmetric dimethylarginine level in mesangial cells through its anti-oxidative properties

被引：17
作者：
Ishibashi, Yuji [1 ]
Matsui, Takanori [1 ]
Ueda, Seiji [2 ]
Fukami, Kei [2 ]
Okuda, Seiya [2 ]
Yamagishi, Sho-ichi [1 ]
机构：
[1] Kurume Univ, Sch Med, Dept Pathophysiol & Therapeut Diabet Vasc Complic, Kurume, Fukuoka 8300011, Japan
[2] Kurume Univ, Sch Med, Dept Med, Kurume, Fukuoka 8300011, Japan
来源：
INTERNATIONAL JOURNAL OF CARDIOLOGY | 2014年 / 176卷 / 03期
关键词：
Irbesartan; AGEs; Oxidative stress; RAGE; DDAH; ADMA; ENDOTHELIAL DYSFUNCTION; INSULIN-RESISTANCE; RECEPTOR; EXPRESSION; MECHANISMS; DISEASE; INJURY;
D O I：
10.1016/j.ijcard.2014.07.299
中图分类号：
R5 [内科学];
学科分类号：
1002 ; 100201 ;
摘要：
[No abstract available]
引用
收藏
页码：1120 / 1122
页数：3
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