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Evaluation of Anti-Inflammatory Activities of Qingre-Qushi Recipe (QRQS) against Atopic Dermatitis: Potential Mechanism of Inhibition of IL-33/ST2 Signal Transduction
被引:12
作者:

Chen, Mengjiao
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机构:
Shanghai Univ Tradit Chinese Med, Dept Dermatol, Shuguang Hosp, Shanghai 200000, Peoples R China Shanghai Univ Tradit Chinese Med, Dept Dermatol, Shuguang Hosp, Shanghai 200000, Peoples R China

Ding, Peijun
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h-index: 0
机构:
Shanghai Univ Tradit Chinese Med, Dept Dermatol, Shuguang Hosp, Shanghai 200000, Peoples R China Shanghai Univ Tradit Chinese Med, Dept Dermatol, Shuguang Hosp, Shanghai 200000, Peoples R China

Yang, Lili
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h-index: 0
机构:
Shanghai Univ Tradit Chinese Med, Dept Dermatol, Shuguang Hosp, Shanghai 200000, Peoples R China Shanghai Univ Tradit Chinese Med, Dept Dermatol, Shuguang Hosp, Shanghai 200000, Peoples R China

He, Xufeng
论文数: 0 引用数: 0
h-index: 0
机构:
Shanghai Univ Tradit Chinese Med, Dept Dermatol, Shuguang Hosp, Shanghai 200000, Peoples R China Shanghai Univ Tradit Chinese Med, Dept Dermatol, Shuguang Hosp, Shanghai 200000, Peoples R China

Gao, Chunjie
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h-index: 0
机构:
Shanghai Univ Tradit Chinese Med, Dept Dermatol, Shuguang Hosp, Shanghai 200000, Peoples R China Shanghai Univ Tradit Chinese Med, Dept Dermatol, Shuguang Hosp, Shanghai 200000, Peoples R China

Yang, Guoxun
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h-index: 0
机构:
Fudan Univ, Sch Pharm, Shanghai 200000, Peoples R China Shanghai Univ Tradit Chinese Med, Dept Dermatol, Shuguang Hosp, Shanghai 200000, Peoples R China

Zhang, Huimin
论文数: 0 引用数: 0
h-index: 0
机构:
Shanghai Univ Tradit Chinese Med, Dept Dermatol, Shuguang Hosp, Shanghai 200000, Peoples R China Shanghai Univ Tradit Chinese Med, Dept Dermatol, Shuguang Hosp, Shanghai 200000, Peoples R China
机构:
[1] Shanghai Univ Tradit Chinese Med, Dept Dermatol, Shuguang Hosp, Shanghai 200000, Peoples R China
[2] Fudan Univ, Sch Pharm, Shanghai 200000, Peoples R China
基金:
中国国家自然科学基金;
关键词:
NF-KAPPA-B;
INFLAMMATORY RESPONSES;
MOUSE MODEL;
EXPRESSION;
DISEASE;
ADULT;
ST2;
D O I:
10.1155/2017/2489842
中图分类号:
R [医药、卫生];
学科分类号:
10 ;
摘要:
To evaluate the anti-inflammatory activities of QRQS against AD and the inhibitory molecular mechanisms of IL-33/ST2 signal transduction, BALB/c mice were divided into six groups (normal control, OVA control, low-dose of QRQS, middle-dose of QRQS, high-dose of QRQS, and cetirizine) and epicutaneously exposed to ovalbumin or PBS for 3 weeks and treated with QRQS for 2 weeks. Skin biopsies and blood samples were obtained for histological study, antibody analysis, and RNA isolation. HaCaT cells, stimulated by TNF-alpha and IFN-gamma, were treated with QRQS to evaluate mRNA and protein expression by RT-PCR and ELISA. QRQS decreased both epidermal and dermal thickness, alleviated dermatitis, and reduced IL-33 and ST2 positive cell numbers. The concentration of specific IgE, IgG, IgG1, and IgG2a antibodies in serum and the expression of IL-33, ST2, IL-1RAcP, IL-4, and IL-13 mRNA in the skin were suppressed. No significant difference exists in TNF-alpha or IFN-gamma QRQS decreased IL-33 mRNA and protein secretion in HaCaT cells exposed to TNF-alpha and IFN-gamma in a time-and concentration-dependent manner. QRQS regulates related molecule expression of ovalbumin-induced dermatitis involved in the IL-33/ST2 signaling axis in the treatment of acute AD.
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页数:11
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机构: IMMUNOBIOL RES INST,ANNANDALE,NJ

HIRSCH, RL
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机构: IMMUNOBIOL RES INST,ANNANDALE,NJ

SCHNEIDER, L
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机构: IMMUNOBIOL RES INST,ANNANDALE,NJ

MOODY, C
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机构: IMMUNOBIOL RES INST,ANNANDALE,NJ

TAKAOKA, R
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机构: IMMUNOBIOL RES INST,ANNANDALE,NJ

LI, SH
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机构: IMMUNOBIOL RES INST,ANNANDALE,NJ

MEYERSON, LA
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机构: IMMUNOBIOL RES INST,ANNANDALE,NJ

MARIAM, SG
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机构: IMMUNOBIOL RES INST,ANNANDALE,NJ

GOLDSTEIN, G
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机构: IMMUNOBIOL RES INST,ANNANDALE,NJ

HANIFIN, JM
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机构: IMMUNOBIOL RES INST,ANNANDALE,NJ