Endoplasmic Reticulum (ER) Stress Induces p44/42 Mitogen-Activated Protein Kinase (MAPK) Signaling, Inflammation and Renin-Angiotensin System (RAS) Activity in Forebrain and Contributes to Sympathetic Nerve Activity in Heart Failure Rats

被引：0

作者：

Wei, Shun-Guang ^{[1
]}

Yu, Yang ^{[1
]}

Weiss, Robert M. ^{[1
]}

Felder, Robert B. ^{[1
,2
]}

机构：

[1] Univ Iowa, Carver Coll Med, Internal Med, Iowa City, IA USA

[2] VA Med Ctr, Iowa City, IA USA

来源：

FASEB JOURNAL | 2016年 / 30卷

关键词：

D O I：

暂无

中图分类号：

Q5 [生物化学]; Q7 [分子生物学];

学科分类号：

071010 ; 081704 ;

摘要：

1005.2

引用

页数：2

共 11 条

[1] Inhibition of Mitogen-Activated Protein Kinase (MAPK) Signaling Ameliorates Endoplasmic Reticulum (ER) Stress, Inflammation and Sympathetic Nerve Activity in Heart Failure Rats.
Wei, Shun-Guang
Yu, Yang
Zhang, Zhi-hua
Weiss, Robert M.
Felder, Robert B.
HYPERTENSION, 2014, 64
[2] Endoplasmic reticulum stress increases brain MAPK signaling, inflammation and renin-angiotensin system activity and sympathetic nerve activity in heart failure
Wei, Shun-Guang
Yu, Yang
Weiss, Robert M.
Felder, Robert B.
AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY, 2016, 311 (04): : H871 - H880
[3] Inhibition of Brain Mitogen-Activated Protein Kinase Signaling Reduces Central Endoplasmic Reticulum Stress and Inflammation and Sympathetic Nerve Activity in Heart Failure Rats
Wei, Shun-Guang
Yu, Yang
Weiss, Robert M.
Felder, Robert B.
HYPERTENSION, 2016, 67 (01) : 229 - 236
[4] p44/42 mitogen-activated protein kinase differentially mediates sympathetic excitation in heart failure rats
Wei, Shun-Guang
Yu, Yang
Zhang, Zhi-Hua
Felder, Robert B.
CIRCULATION, 2006, 114 (18) : 309 - 309
[5] Angiotensin II-triggered p44/42 mitogen-activated protein kinase mediates sympathetic excitation in heart failure rats
Wei, Shun-Guang
Yu, Yang
Zhang, Zhi-Hua
Weiss, Robert M.
Felder, Robert B.
HYPERTENSION, 2008, 52 (02) : 342 - 350
[6] Inhibition of Brain Mitogen-Activated Protein Kinase Signaling Reduces Central Endoplasmic Reticulum Stress and Inflammation and Sympathetic Nerve Activity in Heart Failure Rats (vol 67, pg 229, 2016)
Wei
HYPERTENSION, 2018, 72 (03) : E32 - E32
[7] Brain p44/42 mitogen-activated protein kinase contributes to the sympathetic response to blood-borne TNF-α in rats
Zhang, Zhihua
Yu, Yang
Wei, Shun-Guang
Felder, Robert B.
FASEB JOURNAL, 2010, 24
[8] Confluence of vascular endothelial cells induces cell cycle exit by inhibiting p42/p44 mitogen-activated protein kinase activity
Viñals, F
Pouysségur, J
MOLECULAR AND CELLULAR BIOLOGY, 1999, 19 (04) : 2763 - 2772
[9] Inhibition of p42 and p44 mitogen-activated protein kinase activity by PD48059 does not suppress nerve growth factor-induced survival of sympathetic neurones
Virdee, K
Tolkovsky, AM
JOURNAL OF NEUROCHEMISTRY, 1996, 67 (05) : 1801 - 1805
[10] Silencing Epidermal Growth Factor Receptor Reduces P44/42 Mitogen-Activated Protein Kinase Signaling in the Hypothalamic Paraventricular Nucleus and Ameliorates Sympathetic Excitation in Rats With Heart Failure After Myocardial Infarction
Yu, Yang
Wei, Shun-Guang
Weiss, Robert M.
Felder, Robert B.
HYPERTENSION, 2019, 74

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