Prolyl isomerases and nuclear function

被引:105
作者
Hunter, T [1 ]
机构
[1] Salk Inst Biol Studies, Mol Biol & Virol Lab, La Jolla, CA 92037 USA
关键词
D O I
10.1016/S0092-8674(00)80906-X
中图分类号
Q5 [生物化学]; Q7 [分子生物学];
学科分类号
071010 ; 081704 ;
摘要
[No abstract available]
引用
收藏
页码:141 / 143
页数:3
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共 22 条
[11]   The Drosophila melanogaster dodo (dod) gene, conserved in humans, is functionally interchangeable with the ESS1 cell division gene of Saccharomyces cerevisiae [J].
Maleszka, R ;
Hanes, SD ;
Hackett, RL ;
deCouet, HG ;
Miklos, GLG .
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1996, 93 (01) :447-451
[12]   Differential regulation of c-Myb-induced transcription activation by a phosphorylation site in the negative regulatory domain [J].
Miglarese, MR ;
Richardson, AF ;
Aziz, N ;
Bender, TP .
JOURNAL OF BIOLOGICAL CHEMISTRY, 1996, 271 (37) :22697-22705
[13]   The Myb oncoprotein: Regulating a regulator [J].
Ness, SA .
BIOCHIMICA ET BIOPHYSICA ACTA-REVIEWS ON CANCER, 1996, 1288 (03) :F123-F139
[14]  
OGATA K, 1994, CELL, V79, P539
[15]   CONFIRMATION OF THE EXISTENCE OF A 3RD FAMILY AMONG PEPTIDYL-PROLYL CIS/TRANS ISOMERASES - AMINO-ACID-SEQUENCE AND RECOMBINANT PRODUCTION OF PARVULIN [J].
RAHFELD, JU ;
RUCKNAGEL, KP ;
SCHELBERT, B ;
LUDWIG, B ;
HACKER, J ;
MANN, K ;
FISCHER, G .
FEBS LETTERS, 1994, 352 (02) :180-184
[16]   Structural and functional analysis of the mitotic rotamase Pin1 suggests substrate recognition is phosphorylation dependent [J].
Ranganathan, R ;
Lu, KP ;
Hunter, T ;
Noel, JP .
CELL, 1997, 89 (06) :875-886
[17]   PROTEIN-FOLDING - PROLYL ISOMERASES JOIN THE FOLD [J].
SCHMID, FX .
CURRENT BIOLOGY, 1995, 5 (09) :993-994
[18]   Catalysis of protein folding by parvulin [J].
Scholz, C ;
Rahfeld, J ;
Fischer, G ;
Schmid, FX .
JOURNAL OF MOLECULAR BIOLOGY, 1997, 273 (03) :752-762
[19]   Cyclophilin active site mutants have native prolyl isomerase activity with a protein substrate [J].
Scholz, C ;
Schindler, T ;
Dolinski, K ;
Heitman, J ;
Schmid, FX .
FEBS LETTERS, 1997, 414 (01) :69-73
[20]  
SHEN M, 1998, IN PRESS GENES DEV