Epigenetic Regulation of Inflammatory Signaling and Inflammation-Induced Cancer

被引:28
作者
Tan, Shawn Ying Xuan [1 ]
Zhang, Jieqiong [1 ,2 ]
Tee, Wee-Wei [1 ,2 ,3 ]
机构
[1] ASTAR, Inst Mol & Cell Biol, Chromatin Dynam & Dis Epigenet Lab, Singapore, Singapore
[2] Natl Univ Singapore, Yong Loo Lin Sch Med, Dept Physiol, Singapore, Singapore
[3] Natl Univ Singapore, Yong Loo Lin Sch Med Natl, NUS Ctr Canc Res, Singapore, Singapore
来源
FRONTIERS IN CELL AND DEVELOPMENTAL BIOLOGY | 2022年 / 10卷
基金
新加坡国家研究基金会; 英国医学研究理事会;
关键词
cancer; inflammation; epigenetics; histone modifications; high-order genome organization; super-enhancer; senescence; NF-KAPPA-B; HELICOBACTER-PYLORI INFECTION; HISTONE DEACETYLASE INHIBITOR; GASTRIC EPITHELIAL-CELLS; ABERRANT DNA METHYLATION; GENE-EXPRESSION PROGRAM; SUPER-ENHANCERS; INTERCHROMOSOMAL ASSOCIATIONS; TRANSCRIPTION FACTORS; BROMODOMAIN PROTEIN;
D O I
10.3389/fcell.2022.931493
中图分类号
Q2 [细胞生物学];
学科分类号
071009 ; 090102 ;
摘要
Epigenetics comprise a diverse array of reversible and dynamic modifications to the cell's genome without implicating any DNA sequence alterations. Both the external environment surrounding the organism, as well as the internal microenvironment of cells and tissues, contribute to these epigenetic processes that play critical roles in cell fate specification and organismal development. On the other hand, dysregulation of epigenetic activities can initiate and sustain carcinogenesis, which is often augmented by inflammation. Chronic inflammation, one of the major hallmarks of cancer, stems from proinflammatory cytokines that are secreted by tumor and tumor-associated cells in the tumor microenvironment. At the same time, inflammatory signaling can establish positive and negative feedback circuits with chromatin to modulate changes in the global epigenetic landscape. In this review, we provide an in-depth discussion of the interconnected crosstalk between epigenetics and inflammation, specifically how epigenetic mechanisms at different hierarchical levels of the genome control inflammatory gene transcription, which in turn enact changes within the cell's epigenomic profile, especially in the context of inflammation-induced cancer.
引用
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页数:21
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