Etiopathogenesis of systemic lupus erythematosus

被引：113

作者：

Herrmann, M ^{[1
]}

Voll, RE ^{[1
]}

Kalden, JR ^{[1
]}

机构：

[1] Univ Erlangen Nurnberg, Dept Med 3, Inst Clin Immunol & Rheumatol, D-91054 Erlangen, Germany

来源：

IMMUNOLOGY TODAY | 2000年 / 21卷 / 09期

关键词：

D O I：

10.1016/S0167-5699(00)01675-3

中图分类号：

R392 [医学免疫学]; Q939.91 [免疫学];

学科分类号：

100102 ;

摘要：

Martin Herrmann and colleagues briefly discuss how autoimmunity might be initiated and maintained in the systemic autoimmune disease systemic lupus erythematosus.

引用

页码：424 / 426

页数：3

共 28 条

[1] Dendritic cells acquire antigen from apoptotic cells and induce class I restricted CTLs [J].

Albert, ML ;

Sauter, B ;

Bhardwaj, N .

NATURE, 1998, 392 (6671) :86-89

[2]

Andreassen K, 1999, EUR J IMMUNOL, V29, P2715, DOI 10.1002/(SICI)1521-4141(199909)29:09<2715::AID-IMMU2715>3.0.CO

[3]

2-#

[4] Role of nucleosomes for induction and glomerular binding of autoantibodies in lupus nephritis [J].

Berden, JHM ;

Licht, R ;

van Bruggen, MCJ ;

Tax, WJM .

CURRENT OPINION IN NEPHROLOGY AND HYPERTENSION, 1999, 8 (03) :299-306

[5] Serum amyloid P component controls chromatin degradation and prevents antinuclear autoimmunity [J].

Bickerstaff, MCM ;

Botto, M ;

Hutchinson, WL ;

Herbert, J ;

Tennent, GA ;

Bybee, A ;

Mitchell, DA ;

Cook, HT ;

Butler, PJG ;

Walport, MJ ;

Pepys, MB .

NATURE MEDICINE, 1999, 5 (06) :694-697

[6] Homozygous C1q deficiency causes glomerulonephritis associated with multiple apoptotic bodies [J].

Botto, M ;

Dell'Agnola, C ;

Bygrave, AE ;

Thompson, EM ;

Cook, HT ;

Petry, F ;

Loos, M ;

Pandolfi, PP ;

Walport, MJ .

NATURE GENETICS, 1998, 19 (01) :56-59

[7] Cleavage by granzyme B is strongly predictive of autoantigen status: Implications for initiation of autoimmunity [J].

Casciola-Rosen, L ;

Andrade, F ;

Ulanet, D ;

Wong, WB ;

Rosen, A .

JOURNAL OF EXPERIMENTAL MEDICINE, 1999, 190 (06) :815-825

[8] Uncoupling of immune complex formation and kidney damage in autoimmune glomerulonephritis [J].

Clynes, R ;

Dumitru, C ;

Ravetch, JV .

SCIENCE, 1998, 279 (5353) :1052-1054

[9] STRUCTURE AND SPECIFICITY OF T-CELL RECEPTORS EXPRESSED BY POTENTIALLY PATHOGENIC ANTI-DNA AUTOANTIBODY-INDUCING T-CELLS IN HUMAN LUPUS [J].

DESAIMEHTA, A ;

MAO, CC ;

RAJAGOPALAN, S ;

ROBINSON, T ;

DATTA, SK .

JOURNAL OF CLINICAL INVESTIGATION, 1995, 95 (02) :531-541

[10] Macrophages that have ingested apoptotic cells in vitro inhibit proinflammatory cytokine production through autocrine/paracrine mechanisms involving TGF-β, PGE2, and PAF [J].

Fadok, VA ;

Bratton, DL ;

Konowal, A ;

Freed, PW ;

Westcott, JY ;

Henson, PM .

JOURNAL OF CLINICAL INVESTIGATION, 1998, 101 (04) :890-898

← 1 2 3 →