Plasmacytoid Denditic Cells (pDC) in Multiple Sclerosis: IFN-β Treatment Inhibits pDC Secretion of Chemokines Specific for CCR5-Positive Th1 Cells

被引：0
作者：
Aung, Latt
Dhib-Jalbut, Suhayl
Balashov, Konstantin E.
机构：
[1] TEAGASC, Natl Food Ctr, Dublin 15, Ireland
[2] Univ Estadual Campinas, Paris 16, France
[3] Osaka Univ, Toyonaka, Osaka 560, Japan
[4] Kazakh Acad Sci, Alma Ata 480021, Kazakhstan
来源：
NEUROLOGY | 2010年 / 74卷 / 09期
关键词：
D O I：
暂无
中图分类号：
R74 [神经病学与精神病学];
学科分类号：
摘要：
引用
收藏
页码：A372 / A373
页数：2
相关论文
共 50 条
[21]   IFN-γ Production by Amyloid β-Specific Th1 Cells Promotes Microglial Activation and Increases Plaque Burden in a Mouse Model of Alzheimer's Disease [J].
Browne, Tara C. ;
McQuillan, Keith ;
McManus, Roisin M. ;
O'Reilly, Julie-Ann ;
Mills, Kingston H. G. ;
Lynch, Marina A. .
JOURNAL OF IMMUNOLOGY, 2013, 190 (05) :2241-2251
[22]   Enhanced secretion of IFN-γ by activated Th1 cells occurs via reverse signaling through TNF-related activation-induced cytokine [J].
Chen, NJ ;
Huang, MW ;
Hsieh, SL .
JOURNAL OF IMMUNOLOGY, 2001, 166 (01) :270-276
[23]   IL-10 production by conventional Th1 cells provides negative feedback regulation of IFN-γ secretion during anti-microbial responses [J].
Jankovic, Dragana ;
Kullberg, Marika C. ;
Caspar, Patricia ;
Sher, Alan .
JOURNAL OF IMMUNOLOGY, 2006, 176 :S13-S13
[24]   ifn-γ-dependent secretion of IL-10 from Th1 cells and microglia/macrophages contributes to functional recovery after spinal cord injury [J].
Ishii, H. ;
Tanabe, S. ;
Ueno, M. ;
Kubo, T. ;
Kayama, H. ;
Serada, S. ;
Fujimoto, M. ;
Takeda, K. ;
Naka, T. ;
Yamashita, T. .
CELL DEATH & DISEASE, 2013, 4 :e710-e710
[25]   ifn-γ-dependent secretion of IL-10 from Th1 cells and microglia/macrophages contributes to functional recovery after spinal cord injury [J].
H Ishii ;
S Tanabe ;
M Ueno ;
T Kubo ;
H Kayama ;
S Serada ;
M Fujimoto ;
K Takeda ;
T Naka ;
T Yamashita .
Cell Death & Disease, 2013, 4 :e710-e710
[26]   CTLA-4 Is Preferentially Expressed on Human IL-17 Producing CD4+CCR6+Memory Th17 T Cells Relative to IFN-γ Producing Th1 Cells [J].
Krummey, S. M. ;
Floyd, T. L. ;
Mehta, A. K. ;
Ford, M. L. .
AMERICAN JOURNAL OF TRANSPLANTATION, 2012, 12 :144-144
[27]   Allergen-specific Th1 cells counteract efferent Th2 cell-dependent bronchial hyperresponsiveness and eosinophilic inflammation partly via IFN-γ [J].
Huang, TJ ;
MacAry, PA ;
Eynott, P ;
Moussavi, A ;
Daniel, KC ;
Askenase, PW ;
Kemeny, DM ;
Chung, KF .
JOURNAL OF IMMUNOLOGY, 2001, 166 (01) :207-217
[28]   Invariant NKT Cells Producing IL-4 or IL-10, But Not IFN-γ, Inhibit the Th1 Response in Experimental Autoimmune Encephalomyelitis, Whereas None of These Cells Inhibits the Th17 Response [J].
Oh, Sae Jin ;
Chung, Doo Hyun .
JOURNAL OF IMMUNOLOGY, 2011, 186 (12) :6815-6821
[29]   Indispensable role for TNF-α and IFN-γ at the effector phase of liver injury mediated by Th1 cells specific to hepatitis B virus surface antigen [J].
Ohta, A ;
Sekimoto, M ;
Sato, M ;
Koda, T ;
Nishimura, S ;
Iwakura, Y ;
Sekikawa, K ;
Nishimura, T .
JOURNAL OF IMMUNOLOGY, 2000, 165 (02) :956-961
[30]   Virus overrides the propensity of human CD40L-activated plasmacytoid dendritic cells to produce Th2 mediators through synergistic induction of IFN-γ and Th1 chemokine production [J].
Bendriss-Vermare, N ;
Burg, S ;
Kanzler, H ;
Chaperot, L ;
Duhen, T ;
de Bouteiller, O ;
D'agostini, M ;
Bridon, JM ;
Durand, I ;
Sederstrom, JM ;
Chen, W ;
Plumas, J ;
Jacob, MC ;
Liu, YJ ;
Garrone, P ;
Trinchieri, G ;
Caux, C ;
Brière, F .
JOURNAL OF LEUKOCYTE BIOLOGY, 2005, 78 (04) :954-966
12345