Molecular biology of α-synuclein dementia with Lewy bodies -: Dementia with Lewy bodies

被引:0
作者
Riess, O [1 ]
Krüger, R [1 ]
Kobayashi, H [1 ]
Holzmann, C [1 ]
Hattori, N [1 ]
Mizuno, Y [1 ]
机构
[1] Univ Rostock, Dept Med Genet, D-18055 Rostock, Germany
来源
MAPPING THE PROGRESS OF ALZHEIMER'S AND PARKINSON'S DISEASE | 2002年 / 51卷
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暂无
中图分类号
B84 [心理学]; C [社会科学总论]; Q98 [人类学];
学科分类号
03 ; 0303 ; 030303 ; 04 ; 0402 ;
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页码:497 / 508
页数:12
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共 92 条
[1]   Mice lacking α-synuclein display functional deficits in the nigrostriatal dopamine system [J].
Abeliovich, A ;
Schmitz, Y ;
Fariñas, I ;
Choi-Lundberg, D ;
Ho, WH ;
Castillo, PE ;
Shinsky, N ;
Verdugo, JMG ;
Armanini, M ;
Ryan, A ;
Hynes, M ;
Phillips, H ;
Sulzer, D ;
Rosenthal, A .
NEURON, 2000, 25 (01) :239-252
[2]   PERIPHERAL NERVOUS SYSTEM-SPECIFIC GENES IDENTIFIED BY SUBTRACTIVE CDNA CLONING [J].
AKOPIAN, AN ;
WOOD, JN .
JOURNAL OF BIOLOGICAL CHEMISTRY, 1995, 270 (36) :21264-21270
[3]   Genetic analysis of families with Parkinson disease that carry the Ala53Thr mutation in the gene encoding α-synuclein [J].
Athanassiadou, A ;
Voutsinas, G ;
Psiouri, L ;
Leroy, E ;
Polymeropoulos, MH ;
Ilias, A ;
Maniatis, GM ;
Papapetropoulos, T .
AMERICAN JOURNAL OF HUMAN GENETICS, 1999, 65 (02) :555-558
[4]   Neural expression profile of α-synuclein in developing human cortex [J].
Bayer, TA ;
Jäkälä, P ;
Hartmann, T ;
Egensperger, R ;
Buslei, R ;
Falkai, P ;
Beyreuther, K .
NEUROREPORT, 1999, 10 (13) :2799-2803
[5]   Degradation of α-synuclein by proteasome [J].
Bennett, MC ;
Bishop, JF ;
Leng, Y ;
Chock, PB ;
Chase, TN ;
Mouradian, MM .
JOURNAL OF BIOLOGICAL CHEMISTRY, 1999, 274 (48) :33855-33858
[6]   Parkinson's disease-associated α-sylnuclein is more fibrillogenic than β- and γ-synuclein and cannot cross-seed its homologs [J].
Biere, AL ;
Wood, SJ ;
Wypych, J ;
Steavenson, S ;
Jiang, YJ ;
Anafi, D ;
Jacobsen, FW ;
Jarosinski, MA ;
Wu, GM ;
Louis, JC ;
Martin, F ;
Narhi, LO ;
Citron, M .
JOURNAL OF BIOLOGICAL CHEMISTRY, 2000, 275 (44) :34574-34579
[7]   Accelerated in vitro fibril formation by a mutant α-synuclein linked to early-onset Parkinson disease [J].
Conway, KA ;
Harper, JD ;
Lansbury, PT .
NATURE MEDICINE, 1998, 4 (11) :1318-1320
[8]   OXIDATIVE STRESS, GLUTAMATE, AND NEURODEGENERATIVE DISORDERS [J].
COYLE, JT ;
PUTTFARCKEN, P .
SCIENCE, 1993, 262 (5134) :689-695
[9]   Non-Aβ component of Alzheimer's disease amyloid (NAC) revisited -: NAC and α-synuclein are not associated with Aβ amyloid [J].
Culvenor, JG ;
McLean, CA ;
Cutt, S ;
Campbell, BCV ;
Maher, F ;
Jäkälä, P ;
Hartmann, T ;
Beyreuther, K ;
Masters, CL ;
Li, QX .
AMERICAN JOURNAL OF PATHOLOGY, 1999, 155 (04) :1173-1181
[10]   Wild-type but not Parkinson's disease-related Ala-53 → Thr mutant α-synuclein protects neuronal cells from apoptotic stimuli [J].
da Costa, CA ;
Ancolio, K ;
Checler, F .
JOURNAL OF BIOLOGICAL CHEMISTRY, 2000, 275 (31) :24065-24069