Poststroke-Bipolar Affective Disorder

被引：1

作者：

Bengesser, S. A. ^{[1
]}

Wurm, W. E. ^{[1
]}

Lackner, N. ^{[1
]}

Birner, A. ^{[1
]}

Reininghaus, B. ^{[1
]}

Kapfhammer, H. -P. ^{[1
]}

Reininghaus, E. ^{[1
]}

机构：

[1] Med Univ Graz, Univ Klinikum Psychiat, A-8036 Graz, Austria

来源：

FORTSCHRITTE DER NEUROLOGIE PSYCHIATRIE | 2013年 / 81卷 / 08期

关键词：

chronic inflammation; vascular risk; poststroke mania; secondary mania; GENOME-WIDE ASSOCIATION; WHITE-MATTER HYPERINTENSITIES; HEMISPHERIC CEREBRAL INFARCTION; SUBGENUAL PREFRONTAL CORTEX; SEROTONIN TRANSPORTER GENE; BASAL GANGLIA; FUNCTIONAL NEUROANATOMY; CIRCADIAN GENES; BRAIN INJURY; METAANALYSIS;

D O I：

10.1055/s-0033-1335731

中图分类号：

R74 [神经病学与精神病学];

学科分类号：

摘要：

A few weeks after suffering from a basal ganglia infarction (globus pallidus) with left-sided hemiplegia, a 23-year-old woman exhibited for the first time a pronounced mania with self-endangerment. The use of oral contraceptives was the only determinable risk factor. During the further course, the mother also developed a depressive disorder. Thus a certain genetic predisposition for affective disorders may be relevant, although this would not explain the outbreak by itself. An association between the right-sided basal ganglia infarction and the occurrence of a bipolar affective disorder has been described in the literature. Vascular or, respectively, inflammatory risk factors in synopsis with the aetiopathogenesis of bipolar affective disorders are also discussed in depth in this case report.

引用

页码：459 / 463

页数：5

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