Blockade of RAGE suppresses periodontitis-associated bone loss in diabetic mice

被引:286
作者
Lalla, E
Lamster, IB
Feit, M
Huang, L
Spessot, A
Qu, W
Kislinger, T
Lu, Y
Stern, DM
Schmidt, AM
机构
[1] Columbia Univ, Sch Dent & Oral Surg, Div Periodont, New York, NY 10032 USA
[2] Columbia Univ, Coll Phys & Surg, Dept Physiol, New York, NY 10032 USA
[3] Columbia Univ, Coll Phys & Surg, Dept Surg, New York, NY 10032 USA
[4] Columbia Univ, Coll Phys & Surg, Dept Med, New York, NY 10032 USA
关键词
D O I
10.1172/JCI8942
中图分类号
R-3 [医学研究方法]; R3 [基础医学];
学科分类号
1001 ;
摘要
Diabetes is associated with increased prevalence, severity, and progression of periodontal disease. To test the hypothesis that activation of RAGE (Receptor for Advanced Glycation End products) contributes to the pathogenesis of diabetes-associated periodontitis, we treated diabetic mice, infected with the human periodontal pathogen Porphyromonas gingivalis, with soluble RAGE (sRAGE). sRAGE is the extracellular domain of the receptor, which binds ligand and blocks interaction with, and activation of, cell-surface RAGE. Blockade of RAGE diminished alveolar bone loss in a dose-dependent manner. Moreover, we noted decreased generation of the proinflammatory cytokines TNF-alpha and IL-6 in gingival tissue, as well as decreased levels of matrix metalloproteinases. Gingival AGEs were also reduced in mice treated with sRAGE, paralleling the observed suppression in alveolar bone loss. These findings link RAGE and exaggerated inflammatory responses to the pathogenesis of destructive periodontal disease in diabetes.
引用
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页码:1117 / 1124
页数:8
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