Hypersensitivity Reactions to Nonsteroidal Anti-Inflammatory Drugs

被引：39

作者：

Torres, Maria Jose ^{[1
]}

Barrionuevo, Esther ^{[1
]}

Kowalski, Marek ^{[2
,3
]}

Blanca, Miguel ^{[1
]}

机构：

[1] Univ Hosp IBIMA, Allergy Unit, Malaga 29008, Spain

[2] Med Univ Lodz, Dept Immunol Rheumatol & Allergy, Lodz, Poland

[3] Med Univ Lodz, Dept Immunol Rheumatol & Allergy, Chair Clin Immunol & Microbiol, PL-92213 Lodz, Poland

来源：

IMMUNOLOGY AND ALLERGY CLINICS OF NORTH AMERICA | 2014年 / 34卷 / 03期

关键词：

Nonsteroidal anti-inflammatory drugs; Hypersensitivity; Clinical diagnosis; Allergic; Nonallergic; Management; EXACERBATED RESPIRATORY-DISEASE; ASPIRIN-INDUCED ASTHMA; EXERCISE-INDUCED ANAPHYLAXIS; PARACETAMOL ACETAMINOPHEN HYPERSENSITIVITY; PHOTOALLERGIC CONTACT-DERMATITIS; CHRONIC IDIOPATHIC URTICARIA; PERIPHERAL-BLOOD LEUKOCYTES; LEUKOTRIENE C-4 SYNTHASE; GENOME-WIDE ASSOCIATION; INTOLERANT ASTHMA;

D O I：

10.1016/j.iac.2014.04.001

中图分类号：

R392 [医学免疫学];

学科分类号：

100102 ;

摘要：

Nonsteroidal anti-inflammatory drugs (NSAIDs) are the drugs most commonly involved in hypersensitivity drug reactions. Such reactions can be due to the release of inflammatory mediators in the absence of specific immunologic recognition, or immunoglobulin E (IgE)- or T-cell-selective responses. The former include upper and lower airway symptoms in patients with chronic underlying respiratory disease, the exacerbation of chronic spontaneous urticaria, and the induction of cutaneous symptoms. The latter include selective responses to a single NSAID with good tolerance to strong cyclooxygenase-1 inhibitors, with a putative IgE or T-cell mechanism proposed. These reactions can be acute or delayed.

引用

页码：507 / +

页数：20

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