Single-nucleotide polymorphisms and their role in coronary artery disease: Where do we stand now?

被引：4

作者：

Papageorgiou, Nikolaos ^{[1
]}

Tousoulis, Dimitris ^{[2
]}

机构：

[1] St Bartholomews Hosp, Barts Heart Ctr, London EC1A 7BE, England

[2] Hippokrateion Hosp, Cardiol Dept 1, Athens, Greece

来源：

HELLENIC JOURNAL OF CARDIOLOGY | 2018年 / 59卷 / 01期

关键词：

FIBRINOGEN GENETIC-VARIABILITY; MENDELIAN RANDOMIZATION; MYOCARDIAL-INFARCTION; FATTY LIVER; ACUTE-PHASE; ASSOCIATION; RISK; ATHEROSCLEROSIS; PROTEIN; ERAD;

D O I：

10.1016/j.hjc.2018.02.013

中图分类号：

R5 [内科学];

学科分类号：

1002 ; 100201 ;

摘要：

引用

页码：14 / 15

页数：2

共 13 条

[1] Genetic polymorphism on endothelial nitric oxide synthase affects endothelial activation and inflammatory response during the acute phase of myocardial infarction [J].

Antoniades, C ;

Tousoulis, D ;

Vasiliadou, C ;

Pitsavos, C ;

Chrysochoou, C ;

Panagiotakos, D ;

Tentolouris, C ;

Marinou, K ;

Koumallos, N ;

Stefanadis, C .

JOURNAL OF THE AMERICAN COLLEGE OF CARDIOLOGY, 2005, 46 (06) :1101-1109

[2] Genetic polymorphisms of platelet glycoprotein Ia and the risk for premature myocardial infarction - Effects on the release of sCD40L during the acute phase of premature myocardial infarction [J].

Antoniades, Charalambos ;

Tousoulis, Dimitris ;

Vasiliadou, Carmen ;

Stefanadi, Elli ;

Marinou, Kyriakoula ;

Stefanadis, Christodoulos .

JOURNAL OF THE AMERICAN COLLEGE OF CARDIOLOGY, 2006, 47 (10) :1959-1966

[3] Hrd1 and ER-Associated Protein Degradation, ERAD, Are Critical Elements of the Adaptive ER Stress Response in Cardiac Myocytes [J].

Doroudgar, Shirin ;

Voelkers, Mirko ;

Thuerauf, Donna J. ;

Khan, Mohsin ;

Mohsin, Sadia ;

Respress, Jonathan L. ;

Wang, Wei ;

Gude, Natalie ;

Mueller, Oliver J. ;

Wehrens, Xander H. T. ;

Sussman, Mark A. ;

Glembotski, Christopher C. .

CIRCULATION RESEARCH, 2015, 117 (06) :536-546

[4] Ubiquitination regulates the assembly of VLDL in HepG2 cells and is the committing step of the apoB-100 ERAD pathway [J].

Fisher, Eric A. ;

Khanna, Neeraj A. ;

McLeod, Roger S. .

JOURNAL OF LIPID RESEARCH, 2011, 52 (06) :1170-1180

[5] The unfolded protein response: controlling cell fate decisions under ER stress and beyond [J].

Hetz, Claudio .

NATURE REVIEWS MOLECULAR CELL BIOLOGY, 2012, 13 (02) :89-102

[6] Significant correlations between severe fatty liver and risk factors for metabolic syndrome [J].

Hsiao, Pi-Jung ;

Kuo, Kung-Kai ;

Shin, Shyi-Jang ;

Yang, Yi-Hsin ;

Lin, Wen-Yi ;

Yang, Jeng-Fu ;

Chiu, Chao-Chin ;

Chuang, Wan-Long ;

Tsai, Tong-Rong ;

Yu, Ming-Lung .

JOURNAL OF GASTROENTEROLOGY AND HEPATOLOGY, 2007, 22 (12) :2118-2123

[7] Mendelian randomization suggests no causal association between C-reactive protein and carotid intima-media thickness in the Young Finns study [J].

Kivimaki, Mika ;

Lawlor, Debbie A. ;

Eklund, Carita ;

Smith, George Davey ;

Hurme, Mikko ;

Lehtimaki, Terho ;

Viikari, Jorma S. A. ;

Raitakari, Olli T. .

ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY, 2007, 27 (04) :978-979

[8] Mendelian randomization: Using genes as instruments for making causal inferences in epidemiology [J].

Lawlor, Debbie A. ;

Harbord, Roger M. ;

Sterne, Jonathan A. C. ;

Timpson, Nic ;

Smith, George Davey .

STATISTICS IN MEDICINE, 2008, 27 (08) :1133-1163

[9] Association of nonalcoholic fatty liver disease with insulin resistance [J].

Marchesini, G ;

Brizi, M ;

Morselli-Labate, AM ;

Bianchi, G ;

Bugianesi, E ;

McCullough, AJ ;

Forlani, G ;

Melchionda, N .

AMERICAN JOURNAL OF MEDICINE, 1999, 107 (05) :450-455

[10]

Papageorgiou N, 2017, REV ESP CARDIOL, V70, P34, DOI [10.1016/j.recesp.2016.05.027, 10.1016/j.rec.2016.05.024]

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