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RIG-1 receptor expression in the pathology of Alzheimer's disease
被引:27
作者:

Vaccari, Juan Pablo de Rivero
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Univ Miami, Miller Sch Med, Dept Neurol Surg, Miami, FL 33136 USA
Univ Miami, Miller Sch Med, Miami Project Cure Paralysis, Miami, FL 33136 USA Univ Miami, Miller Sch Med, Dept Neurol Surg, Miami, FL 33136 USA

Brand, Frank J., III
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Univ Miami, Miller Sch Med, Dept Physiol & Biophys, Miami, FL 33136 USA Univ Miami, Miller Sch Med, Dept Neurol Surg, Miami, FL 33136 USA

Sedaghat, Christina
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h-index: 0
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Univ Miami, Miller Sch Med, Miami Project Cure Paralysis, Miami, FL 33136 USA Univ Miami, Miller Sch Med, Dept Neurol Surg, Miami, FL 33136 USA

Mash, Deborah C.
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h-index: 0
机构:
Univ Miami, Miller Sch Med, Dept Neurol, Miami, FL 33136 USA
Univ Miami, Miller Sch Med, Dept Mol & Cellular Pharmacol, Miami, FL 33136 USA Univ Miami, Miller Sch Med, Dept Neurol Surg, Miami, FL 33136 USA

Dietrich, W. Dalton
论文数: 0 引用数: 0
h-index: 0
机构:
Univ Miami, Miller Sch Med, Dept Neurol Surg, Miami, FL 33136 USA
Univ Miami, Miller Sch Med, Miami Project Cure Paralysis, Miami, FL 33136 USA Univ Miami, Miller Sch Med, Dept Neurol Surg, Miami, FL 33136 USA

Keane, Robert W.
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h-index: 0
机构:
Univ Miami, Miller Sch Med, Dept Physiol & Biophys, Miami, FL 33136 USA
Univ Miami, Miller Sch Med, Miami Project Cure Paralysis, Miami, FL 33136 USA Univ Miami, Miller Sch Med, Dept Neurol Surg, Miami, FL 33136 USA
机构:
[1] Univ Miami, Miller Sch Med, Dept Neurol Surg, Miami, FL 33136 USA
[2] Univ Miami, Miller Sch Med, Dept Physiol & Biophys, Miami, FL 33136 USA
[3] Univ Miami, Miller Sch Med, Dept Neurol, Miami, FL 33136 USA
[4] Univ Miami, Miller Sch Med, Dept Mol & Cellular Pharmacol, Miami, FL 33136 USA
[5] Univ Miami, Miller Sch Med, Miami Project Cure Paralysis, Miami, FL 33136 USA
关键词:
Innate immunity;
Rig signaling;
RLR;
Inflammation;
Alzheimer's disease;
Mild cognitive impairment;
MILD COGNITIVE IMPAIRMENT;
AMYLOID PRECURSOR PROTEIN;
INNATE IMMUNE-SYSTEM;
BETA-PEPTIDE;
INFLAMMATION;
ACTIVATION;
POPULATION;
ASTROCYTES;
MECHANISM;
CYTOKINES;
D O I:
10.1186/1742-2094-11-67
中图分类号:
R392 [医学免疫学];
Q939.91 [免疫学];
学科分类号:
100102 ;
摘要:
Background: Neuroinflammation plays a critical role in the pathogenesis of Alzheimer's disease (AD) and involves activation of the innate immune response via recognition of diverse stimuli by pattern recognition receptors (PRRs). The inflammatory inducers and precise innate signaling pathway contributing to AD pathology remain largely undefined. Results: In the present study we analyzed expression levels of innate immune proteins in temporal and occipital cortices from preclinical (no cognitive impairment, NCI, N = 22) to mild cognitive impairment (MCI, N = 20) associated with AD pathology (N = 20) and AD patients (N = 23). We found that retinoic acid-inducible gene-I (RIG-1) is significantly elevated in the temporal cortex and plasma in patients with MCI. In addition, primary human astrocytes stimulated with the RIG-1 ligand 5'ppp RNA showed increased expression of amyloid precursor protein (APP) and amyloid-beta (A beta), supporting the idea that RIG-1 is involved in the pathology of MCI associated with early progression to AD. Conclusion: These findings suggest that RIG-1 may play a critical role in incipient AD.
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