Diabetes and advanced glycoxidation end products

[1] Achmad TH, 1997, METHOD FIND EXP CLIN, V19, P153

[2] Advanced glycation endproducts - role in pathology of diabetic complications [J]. DIABETES RESEARCH AND CLINICAL PRACTICE, 2005, 67 (01) : 3 - 21

[3] DIMINISHED ARTERIAL ELASTICITY IN DIABETES - ASSOCIATION WITH FLUORESCENT ADVANCED GLYCOSYLATION END-PRODUCTS IN COLLAGEN [J]. CARDIOVASCULAR RESEARCH, 1993, 27 (06) : 942 - 945

[4] Inhibition of advanced glycation endproduct formation by acetaldehyde: Role in the cardioprotective effect of ethanol [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1999, 96 (05) : 2385 - 2390

[5] Production of Nε-(carboxymethyl)lysine is impaired in mice deficient in NADPH oxidase -: A role for phagocyte-derived oxidants in the formation of advanced glycation end products during inflammation [J]. DIABETES, 2003, 52 (08) : 2137 - 2143

[6] The myeloperoxidase system of human phagocytes generates Nε-(carboxymethyl)lysine on proteins:: a mechanism for producing advances glycation end products at sites of inflammation [J]. JOURNAL OF CLINICAL INVESTIGATION, 1999, 104 (01) : 103 - 113

[7] [Anonymous], AM J PHYSIOL-ENDOC M

[8] How hyperglycemia promotes atherosclerosis: molecular mechanisms [J]. CARDIOVASCULAR DIABETOLOGY, 2002, 1 (1)

[9] Prevention of incipient diabetic nephropathy by high-dose thiamine and benfotiamine [J]. DIABETES, 2003, 52 (08) : 2110 - 2120

[10] Baliga B S, 2000, Endocr Pract, V6, P435

[1] Achmad TH, 1997, METHOD FIND EXP CLIN, V19, P153

[2] Advanced glycation endproducts - role in pathology of diabetic complications [J]. DIABETES RESEARCH AND CLINICAL PRACTICE, 2005, 67 (01) : 3 - 21

[3] DIMINISHED ARTERIAL ELASTICITY IN DIABETES - ASSOCIATION WITH FLUORESCENT ADVANCED GLYCOSYLATION END-PRODUCTS IN COLLAGEN [J]. CARDIOVASCULAR RESEARCH, 1993, 27 (06) : 942 - 945

[4] Inhibition of advanced glycation endproduct formation by acetaldehyde: Role in the cardioprotective effect of ethanol [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1999, 96 (05) : 2385 - 2390

[5] Production of Nε-(carboxymethyl)lysine is impaired in mice deficient in NADPH oxidase -: A role for phagocyte-derived oxidants in the formation of advanced glycation end products during inflammation [J]. DIABETES, 2003, 52 (08) : 2137 - 2143

[6] The myeloperoxidase system of human phagocytes generates Nε-(carboxymethyl)lysine on proteins:: a mechanism for producing advances glycation end products at sites of inflammation [J]. JOURNAL OF CLINICAL INVESTIGATION, 1999, 104 (01) : 103 - 113

[7] [Anonymous], AM J PHYSIOL-ENDOC M

[8] How hyperglycemia promotes atherosclerosis: molecular mechanisms [J]. CARDIOVASCULAR DIABETOLOGY, 2002, 1 (1)

[9] Prevention of incipient diabetic nephropathy by high-dose thiamine and benfotiamine [J]. DIABETES, 2003, 52 (08) : 2110 - 2120

[10] Baliga B S, 2000, Endocr Pract, V6, P435