Hypereosinophilic syndrome variants: diagnostic and therapeutic considerations

被引：49

作者：

Roufosse, Florence ^{[1
,2
]}

机构：

[1] Univ Libre Bruxelles, Dept Internal Med, Hop Erasme, Brussels, Belgium

[2] Univ Libre Bruxelles, Inst Med Immunol, Gosselies, Belgium

来源：

HAEMATOLOGICA-THE HEMATOLOGY JOURNAL | 2009年 / 94卷 / 09期

关键词：

CHRONIC EOSINOPHILIC LEUKEMIA; LYMPHOCYTIC VARIANT; FIP1L1-PDGFRA FUSION; T-CELLS; IMATINIB; INFLAMMATION; PREVALENCE; CLONALITY; KINASE; GENE;

D O I：

10.3324/haematol.2009.010421

中图分类号：

R5 [内科学];

学科分类号：

1002 ; 100201 ;

摘要：

引用

页码：1188 / 1193

页数：6

共 30 条

[1] Resolution of left and right ventricular thrombosis secondary to hypereosinophilic syndrome (lymphoproliferative variant) with reduced intensity conditioning allogenic stem cell transplantation [J].

Bergua, Juan M. ;

Prieto-Pliego, Elena ;

Roman-Barbera, Alejandro ;

Garcia-Toron, Jose ;

Gomez-Barrado, Jose Javier ;

Marcos, Gonzalo ;

Lopez-Gomez, Luis ;

Tomas, Jose F. .

ANNALS OF HEMATOLOGY, 2008, 87 (11) :937-938

[2] Powerful strategy for polymerase chain reaction-based clonality assessment in T-cell malignancies Report of the BIOMED-2 Concerted Action BHM4 CT98-3936 [J].

Brueggemann, M. ;

White, H. ;

Gaulard, P. ;

Garcia-Sanz, R. ;

Gameiro, P. ;

Oeschger, S. ;

Jasani, B. ;

Ott, M. ;

Delsol, G. ;

Orfao, A. ;

Tiemann, M. ;

Herbst, H. ;

Langerak, A. W. ;

Spaargaren, M. ;

Moreau, E. ;

Groenen, P. J. T. A. ;

Sambade, C. ;

Foroni, L. ;

Carter, G. I. ;

Hummel, M. ;

Bastard, C. ;

Davi, F. ;

Delfau-Larue, M. -H. ;

Kneba, M. ;

van Dongen, J. J. M. ;

Beldjord, K. ;

Molina, T. J. .

LEUKEMIA, 2007, 21 (02) :215-221

[3] HYPEREOSINOPHILIC SYNDROME - ANALYSIS OF 14 CASES WITH REVIEW OF LITERATURE [J].

CHUSID, MJ ;

DALE, DC ;

WEST, BC ;

WOLFF, SM .

MEDICINE, 1975, 54 (01) :1-27

[4] BRIEF REPORT - CLONAL PROLIFERATION OF TYPE-2 HELPER T-CELLS IN A MAN WITH THE HYPEREOSINOPHILIC SYNDROME [J].

COGAN, E ;

SCHANDENE, L ;

CRUSIAUX, A ;

COCHAUX, P ;

VELU, T ;

GOLDMAN, M .

NEW ENGLAND JOURNAL OF MEDICINE, 1994, 330 (08) :535-538

[5] A tyrosine kinase created by fusion of the PDGFRA and FIP1L1 genes as a therapeutic target of imatinib in idiopathic hypereosinophilic syndrome [J].

Cools, J ;

DeAngelo, DJ ;

Gotlib, J ;

Stover, EH ;

Legare, RD ;

Cortes, J ;

Kutok, J ;

Clark, J ;

Galinsky, I ;

Griffin, JD ;

Cross, NCP ;

Tefferi, A ;

Malone, J ;

Alam, R ;

Schrier, SL ;

Schmid, J ;

Rose, M ;

Vandenberghe, P ;

Verhoef, G ;

Boogaerts, M ;

Wlodarska, I ;

Kantarjian, H ;

Marynen, P ;

Coutre, SE ;

Stone, R ;

Gilliland, DG .

NEW ENGLAND JOURNAL OF MEDICINE, 2003, 348 (13) :1201-1214

[6] High serum thymus and activation-regulated chemokine levels in the lymphocytic variant of the hypereosinophilic syndrome [J].

de Lavareille, A ;

Roufosse, F ;

Schmid-Grendelmeier, P ;

Roumier, AS ;

Schandené, L ;

Cogan, E ;

Simon, HU ;

Goldman, M .

JOURNAL OF ALLERGY AND CLINICAL IMMUNOLOGY, 2002, 110 (03) :476-479

[7]

de Lavareille A, 2001, EUR J IMMUNOL, V31, P1037, DOI 10.1002/1521-4141(200104)31:4<1037::AID-IMMU1037>3.0.CO

[8]

2-#

[9]

Galimberti S, 2007, CLIN EXP RHEUMATOL, V25, P17

[10] Treatment of hypereosinophilic syndrome with imatinib mesilate [J].

Gleich, GJ ;

Leiferman, KM ;

Pardanani, A ;

Tefferi, A ;

Butterfield, JH .

LANCET, 2002, 359 (9317) :1577-1578

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