Unzipping RyR2 in adult cardiomyocytes: Getting closer to mechanisms of inherited ventricular arrhythmias?

被引：5
作者：
Femandez-Velasco, Maria [1 ]
Gomez, Ana Maria [1 ]
Richard, Sylvain [1 ]
机构：
[1] Univ Montpellier 1, CHU Arnaud Villeneuve, INSERM, U637, F-34295 Montpellier 5, France
来源：
CARDIOVASCULAR RESEARCH | 2006年 / 70卷 / 03期
关键词：
D O I：
10.1016/j.cardiores.2006.04.007
中图分类号：
R5 [内科学];
学科分类号：
1002 ; 100201 ;
摘要：
引用
收藏
页码：407 / 409
页数：3
相关论文
共 21 条
[1]   Bidirectional ventricular tachycardia and fibrillation elicited in a knock-in mouse model carrier of a mutation in the cardiac ryanodine receptor [J].
Cerrone, M ;
Colombi, B ;
Santoro, M ;
di Barletta, MR ;
Scelsi, M ;
Villani, L ;
Napolitano, C ;
Priori, SG .
CIRCULATION RESEARCH, 2005, 96 (10) :E77-E82
[2]   The control of sarcoplasmic reticulum Ca content in cardiac muscle [J].
Díaz, ME ;
Graham, HK ;
O'Neill, SC ;
Trafford, AW ;
Eisner, DA .
CELL CALCIUM, 2005, 38 (3-4) :391-396
[3]   Ca2+-dependent reduction of IK1 in rat ventricular cells:: A novel paradigm for arrhythmia in heart failure? [J].
Fauconnier, J ;
Lacampagne, A ;
Rauzier, JM ;
Vassort, G ;
Richard, S .
CARDIOVASCULAR RESEARCH, 2005, 68 (02) :204-212
[4]   Arrhythmogenic mutation-linked defects in ryanodine receptor autoregulation reveal a novel mechanism of Ca2+ release channel dysfunction [J].
George, CH ;
Jundi, H ;
Walters, N ;
Thomas, NL ;
West, RR ;
Lai, FA .
CIRCULATION RESEARCH, 2006, 98 (01) :88-97
[5]   Ryanodine receptor mutations associated with stress-induced ventricular tachycardia mediate increased calcium release in stimulated cardiomyocytes [J].
George, CH ;
Higgs, GV ;
Lai, FA .
CIRCULATION RESEARCH, 2003, 93 (06) :531-540
[6]   FKBP12.6 overexpression decreases Ca2+ spark amplitude but enhances [Ca2+]i transient in rat cardiac myocytes [J].
Gómez, AM ;
Schuster, I ;
Fauconnier, J ;
Prestle, J ;
Hasenfuss, G ;
Richard, S .
AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY, 2004, 287 (05) :H1987-H1993
[7]   Regulation of calcium release by interdomain interaction within ryanodine receptors [J].
Ikemoto, N ;
Yamamoto, T .
FRONTIERS IN BIOSCIENCE-LANDMARK, 2002, 7 :D671-D683
[8]   Enhanced basal activity of a cardiac Ca2+ release channel (Ryanodine receptor) mutant associated with ventricular tachycardia and sudden death [J].
Jiang, DW ;
Xiao, BL ;
Zhang, L ;
Chen, SRW .
CIRCULATION RESEARCH, 2002, 91 (03) :218-225
[9]   Enhanced store overload-induced Ca2+ release and channel sensitivity to luminal Ca2+ activation are common defects of RyR2 mutations linked to ventricular tachycardia and sudden death [J].
Jiang, DW ;
Wang, RW ;
Xiao, BL ;
Kong, HH ;
Hunt, DJ ;
Choi, P ;
Zhang, L ;
Chen, SRW .
CIRCULATION RESEARCH, 2005, 97 (11) :1173-1181
[10]   Susceptibility genes and modifiers for cardiac arrhythmias [J].
Kääb, S ;
Schulze-Bahr, E .
CARDIOVASCULAR RESEARCH, 2005, 67 (03) :397-413
123