共 43 条
Leptin-mediated regulation of ICAM-1 is Rho/ROCK dependent and enhances gastric cancer cell migration
被引:45
作者:

Dong, Z.
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机构:
Shandong Univ, Qilu Hosp, Dept Clin Lab, Jinan 250012, Shandong, Peoples R China Shandong Univ, Qilu Hosp, Dept Clin Lab, Jinan 250012, Shandong, Peoples R China

Fu, S.
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h-index: 0
机构:
Shandong Univ, Qilu Hosp, Dept Clin Lab, Jinan 250012, Shandong, Peoples R China Shandong Univ, Qilu Hosp, Dept Clin Lab, Jinan 250012, Shandong, Peoples R China

Xu, X.
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h-index: 0
机构:
Shandong Univ, Qilu Hosp, Dept Obstet & Gynecol, Jinan 250012, Shandong, Peoples R China Shandong Univ, Qilu Hosp, Dept Clin Lab, Jinan 250012, Shandong, Peoples R China

Yang, Y.
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h-index: 0
机构:
Shandong Univ, Qilu Hosp, Dept Clin Lab, Jinan 250012, Shandong, Peoples R China Shandong Univ, Qilu Hosp, Dept Clin Lab, Jinan 250012, Shandong, Peoples R China

Du, L.
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h-index: 0
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Shandong Univ, Qilu Hosp, Dept Clin Lab, Jinan 250012, Shandong, Peoples R China Shandong Univ, Qilu Hosp, Dept Clin Lab, Jinan 250012, Shandong, Peoples R China

Li, W.
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Shandong Univ, Qilu Hosp, Dept Clin Lab, Jinan 250012, Shandong, Peoples R China Shandong Univ, Qilu Hosp, Dept Clin Lab, Jinan 250012, Shandong, Peoples R China

Kan, S.
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Shandong Univ, Qilu Hosp, Dept Clin Lab, Jinan 250012, Shandong, Peoples R China Shandong Univ, Qilu Hosp, Dept Clin Lab, Jinan 250012, Shandong, Peoples R China

Li, Z.
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h-index: 0
机构:
Shandong Univ, Qilu Hosp, Dept Clin Lab, Jinan 250012, Shandong, Peoples R China Shandong Univ, Qilu Hosp, Dept Clin Lab, Jinan 250012, Shandong, Peoples R China

Zhang, X.
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Shandong Univ, Qilu Hosp, Dept Clin Lab, Jinan 250012, Shandong, Peoples R China Shandong Univ, Qilu Hosp, Dept Clin Lab, Jinan 250012, Shandong, Peoples R China

Wang, L.
论文数: 0 引用数: 0
h-index: 0
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Shandong Univ, Qilu Hosp, Dept Clin Lab, Jinan 250012, Shandong, Peoples R China Shandong Univ, Qilu Hosp, Dept Clin Lab, Jinan 250012, Shandong, Peoples R China

Li, J.
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h-index: 0
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Shandong Univ, Qilu Hosp, Dept Clin Lab, Jinan 250012, Shandong, Peoples R China Shandong Univ, Qilu Hosp, Dept Clin Lab, Jinan 250012, Shandong, Peoples R China

Liu, H.
论文数: 0 引用数: 0
h-index: 0
机构:
Shandong Univ, Qilu Hosp, Dept Clin Lab, Jinan 250012, Shandong, Peoples R China Shandong Univ, Qilu Hosp, Dept Clin Lab, Jinan 250012, Shandong, Peoples R China

Qu, X.
论文数: 0 引用数: 0
h-index: 0
机构:
Shandong Univ, Qilu Hosp, Inst Basic Med Sci, Jinan 250012, Shandong, Peoples R China Shandong Univ, Qilu Hosp, Dept Clin Lab, Jinan 250012, Shandong, Peoples R China

Wang, C.
论文数: 0 引用数: 0
h-index: 0
机构:
Shandong Univ, Qilu Hosp, Dept Clin Lab, Jinan 250012, Shandong, Peoples R China Shandong Univ, Qilu Hosp, Dept Clin Lab, Jinan 250012, Shandong, Peoples R China
机构:
[1] Shandong Univ, Qilu Hosp, Dept Clin Lab, Jinan 250012, Shandong, Peoples R China
[2] Shandong Univ, Qilu Hosp, Dept Obstet & Gynecol, Jinan 250012, Shandong, Peoples R China
[3] Shandong Univ, Qilu Hosp, Inst Basic Med Sci, Jinan 250012, Shandong, Peoples R China
基金:
高等学校博士学科点专项科研基金;
关键词:
gastric cancer;
leptin;
ICAM-1;
Rho/ROCK;
migration;
INTERCELLULAR-ADHESION MOLECULE-1;
CLINICAL-SIGNIFICANCE;
HELICOBACTER-PYLORI;
KINASE PATHWAY;
UP-REGULATION;
SERUM-LEVELS;
LUNG-CANCER;
RHO-GTPASES;
EXPRESSION;
RECEPTOR;
D O I:
10.1038/bjc.2014.70
中图分类号:
R73 [肿瘤学];
学科分类号:
100214 ;
摘要:
Background: Our previous study indicates that leptin enhances gastric cancer (GC) invasion. However, the exact effect of leptin on GC metastasis and its underlying mechanism remain unclear. Intercellular adhesion molecule-1 (ICAM-1), a major molecule in stabilising cell-cell and cell-extracellular matrix interactions, is overexpressed and has crucial roles in tumour metastasis. Methods: Here, we investigated leptin and ICAM-1 expression in GC tissues. Furthermore, we characterised the influence of leptin on ICAM-1 expression in GC cells and elucidated the underlying mechanism. Results: Leptin and ICAM-1 were overexpressed in GC tissues, and a strong positive correlation was observed. They were also related with clinical stage or lymph node metastasis. Furthermore, leptin induced GC cell (AGS and MKN-45) migration by upregulating ICAM-1, and knockdown of ICAM-1 by small interference RNA (siRNA) blocked this process. Cell surface ICAM-1, as well as soluble ICAM-1 (sICAM-1), was also enhanced by leptin. Moreover, leptin increased ICAM-1 expression through Rho/ROCK pathway, which was attenuated by pharmacological inhibition of Rho (C3 transferase) or its downstream effector kinase Rho-associated protein kinase (ROCK) (Y-27632). Conclusions: Our findings indicate that leptin enhances GC cell migration by increasing ICAM-1 through Rho/ROCK pathway, which might provide new insight into the significance of leptin in GC.
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收藏
页码:1801 / 1810
页数:10
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Sithu, S.
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Univ Louisville, Sch Med, Dept Physiol & Biophys, Louisville, KY 40292 USA Univ Louisville, Sch Med, Dept Physiol & Biophys, Louisville, KY 40292 USA

Dean, W.
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Univ Louisville, Sch Med, Dept Biochem & Mol Biol, Louisville, KY 40292 USA Univ Louisville, Sch Med, Dept Physiol & Biophys, Louisville, KY 40292 USA

D'Souza, S.
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Univ Louisville, Sch Med, Dept Physiol & Biophys, Louisville, KY 40292 USA Univ Louisville, Sch Med, Dept Physiol & Biophys, Louisville, KY 40292 USA