ATG16L1 and NOD2 Interact in an Autophagy-Dependent Anti-Bacterial Pathway Implicated in Crohn's Disease Pathogenesis

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作者
Homer, Craig R.
Richmond, Amy L.
Rebert, Nancy A.
Achkar, Jean-Paul
McDonald, Christine
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R57 [消化系及腹部疾病];
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W1239
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页码:S681 / S681
页数:1
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[21]   ATG16L1 Contribution to Crohn's Disease Risk in Sicily [J].
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