Signaling at the slit diaphragm

[1] A novel protein, densin, expressed by glomerular podocytes [J]. JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY, 2003, 14 (07): : 1731 - 1737

[2] Genetic models: clues for understanding the pathogenesis of idiopathic nephrotic syndrome [J]. JOURNAL OF CLINICAL INVESTIGATION, 2002, 109 (04) : 447 - 449

[3] Artero RD, 2001, DEVELOPMENT, V128, P4251

[4] The Wiskott-Aldrich syndrome protein acts downstream of CD2 and the CD2AP and PSTPIP1 adaptors to promote formation of the immunological synapse [J]. IMMUNITY, 2003, 18 (01) : 141 - 154

[5] Roles of SLC/CCL21 and CCR7 in human kidney for mesangial proliferation, migration, apoptosis, and tissue homeostasis [J]. JOURNAL OF IMMUNOLOGY, 2002, 168 (09) : 4301 - 4307

[6] Nephrin and Neph1 co-localize at the podocyte foot process intercellular junction and form cis hetero-oligomers [J]. JOURNAL OF BIOLOGICAL CHEMISTRY, 2003, 278 (21) : 19266 - 19271

[7] Bour BA, 2000, GENE DEV, V14, P1498

[8] NPHS2, encoding the glomerular protein podocin, is mutated in autosomal recessive steroid-resistant nephrotic syndrome [J]. NATURE GENETICS, 2000, 24 (04) : 349 - 354

[9] MOLECULAR-BASIS OF PROTEINURIA OF GLOMERULAR ORIGIN [J]. NEW ENGLAND JOURNAL OF MEDICINE, 1978, 298 (15) : 826 - 833

[10] GLOMERULAR PERMSELECTIVITY - BARRIER FUNCTION BASED ON DISCRIMINATION OF MOLECULAR-SIZE AND CHARGE [J]. AMERICAN JOURNAL OF PHYSIOLOGY, 1978, 234 (06): : F455 - F460

[1] A novel protein, densin, expressed by glomerular podocytes [J]. JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY, 2003, 14 (07): : 1731 - 1737

[2] Genetic models: clues for understanding the pathogenesis of idiopathic nephrotic syndrome [J]. JOURNAL OF CLINICAL INVESTIGATION, 2002, 109 (04) : 447 - 449

[3] Artero RD, 2001, DEVELOPMENT, V128, P4251

[4] The Wiskott-Aldrich syndrome protein acts downstream of CD2 and the CD2AP and PSTPIP1 adaptors to promote formation of the immunological synapse [J]. IMMUNITY, 2003, 18 (01) : 141 - 154

[5] Roles of SLC/CCL21 and CCR7 in human kidney for mesangial proliferation, migration, apoptosis, and tissue homeostasis [J]. JOURNAL OF IMMUNOLOGY, 2002, 168 (09) : 4301 - 4307

[6] Nephrin and Neph1 co-localize at the podocyte foot process intercellular junction and form cis hetero-oligomers [J]. JOURNAL OF BIOLOGICAL CHEMISTRY, 2003, 278 (21) : 19266 - 19271

[7] Bour BA, 2000, GENE DEV, V14, P1498

[8] NPHS2, encoding the glomerular protein podocin, is mutated in autosomal recessive steroid-resistant nephrotic syndrome [J]. NATURE GENETICS, 2000, 24 (04) : 349 - 354

[9] MOLECULAR-BASIS OF PROTEINURIA OF GLOMERULAR ORIGIN [J]. NEW ENGLAND JOURNAL OF MEDICINE, 1978, 298 (15) : 826 - 833

[10] GLOMERULAR PERMSELECTIVITY - BARRIER FUNCTION BASED ON DISCRIMINATION OF MOLECULAR-SIZE AND CHARGE [J]. AMERICAN JOURNAL OF PHYSIOLOGY, 1978, 234 (06): : F455 - F460