Inflammatory cytokines, vascular function, and hypertension

[1] Tumor necrosis factor-α-induced hypertension in pregnant rats results in decreased renal neuronal nitric oxide synthase expression [J]. AMERICAN JOURNAL OF HYPERTENSION, 2002, 15 (02) : 170 - 175

[2] Endothelin type A receptor blockade attenuates the hypertension in response to chronic reductions in uterine perfusion pressure [J]. HYPERTENSION, 2001, 37 (02) : 485 - 489

[3] Effect of angiotensin II synthesis blockade on the hypertensive response to chronic reductions in uterine perfusion pressure in pregnant rats [J]. HYPERTENSION, 2001, 38 (03) : 742 - 745

[4] Conrad KP, 1998, AM J REPROD IMMUNOL, V40, P102

[5] Conrad KP, 1997, AM J REPROD IMMUNOL, V37, P240

[6] Reduced endothelial NO-cGMP vascular relaxation pathway during TNF-α-induced hypertension in pregnant rats [J]. AMERICAN JOURNAL OF PHYSIOLOGY-REGULATORY INTEGRATIVE AND COMPARATIVE PHYSIOLOGY, 2002, 282 (02) : R390 - R399

[7] Inflammation and restenosis: implications for therapy [J]. ANNALS OF MEDICINE, 2003, 35 (07) : 523 - 531

[8] TNF-α enhances contraction and inhibits endothelial NO-cGMP relaxation in systemic vessels of pregnant rats [J]. AMERICAN JOURNAL OF PHYSIOLOGY-REGULATORY INTEGRATIVE AND COMPARATIVE PHYSIOLOGY, 2002, 283 (01) : R130 - R143

[9] Pathophysiology of preeclampsia: Linking placental ischemia/hypoxia with microvascular dysfunction [J]. MICROCIRCULATION, 2002, 9 (03) : 147 - 160

[10] Pathophysiology of hypertension during preeclampsia linking placental ischemia with endothelial dysfunction [J]. HYPERTENSION, 2001, 38 (03) : 718 - 722

[1] Tumor necrosis factor-α-induced hypertension in pregnant rats results in decreased renal neuronal nitric oxide synthase expression [J]. AMERICAN JOURNAL OF HYPERTENSION, 2002, 15 (02) : 170 - 175

[2] Endothelin type A receptor blockade attenuates the hypertension in response to chronic reductions in uterine perfusion pressure [J]. HYPERTENSION, 2001, 37 (02) : 485 - 489

[3] Effect of angiotensin II synthesis blockade on the hypertensive response to chronic reductions in uterine perfusion pressure in pregnant rats [J]. HYPERTENSION, 2001, 38 (03) : 742 - 745

[4] Conrad KP, 1998, AM J REPROD IMMUNOL, V40, P102

[5] Conrad KP, 1997, AM J REPROD IMMUNOL, V37, P240

[6] Reduced endothelial NO-cGMP vascular relaxation pathway during TNF-α-induced hypertension in pregnant rats [J]. AMERICAN JOURNAL OF PHYSIOLOGY-REGULATORY INTEGRATIVE AND COMPARATIVE PHYSIOLOGY, 2002, 282 (02) : R390 - R399

[7] Inflammation and restenosis: implications for therapy [J]. ANNALS OF MEDICINE, 2003, 35 (07) : 523 - 531

[8] TNF-α enhances contraction and inhibits endothelial NO-cGMP relaxation in systemic vessels of pregnant rats [J]. AMERICAN JOURNAL OF PHYSIOLOGY-REGULATORY INTEGRATIVE AND COMPARATIVE PHYSIOLOGY, 2002, 283 (01) : R130 - R143

[9] Pathophysiology of preeclampsia: Linking placental ischemia/hypoxia with microvascular dysfunction [J]. MICROCIRCULATION, 2002, 9 (03) : 147 - 160

[10] Pathophysiology of hypertension during preeclampsia linking placental ischemia with endothelial dysfunction [J]. HYPERTENSION, 2001, 38 (03) : 718 - 722