Postnatal Inflammation in the Pathogenesis of Bronchopulmonary Dysplasia

被引：78

作者：

Bhandari, Vineet ^{[1
]}

机构：

[1] Yale Univ, Sch Med, Div Perinatal Med, Dept Pediat, New Haven, CT 06520 USA

来源：

BIRTH DEFECTS RESEARCH PART A-CLINICAL AND MOLECULAR TERATOLOGY | 2014年 / 100卷 / 03期

关键词：

invasive ventilation; infection; hyperoxia; lung; newborn; INDUCED LUNG INJURY; ENDOTHELIAL GROWTH-FACTOR; NEWBORN RAT LUNG; TIDAL VOLUME VENTILATION; POSITIVE AIRWAY PRESSURE; ATTENUATES PULMONARY INFLAMMATION; DEFICIENCY CONFERS PROTECTION; RESPIRATORY-DISTRESS-SYNDROME; MIGRATION INHIBITORY FACTOR; EXTREMELY PREMATURE BABOONS;

D O I：

10.1002/bdra.23220

中图分类号：

Q [生物科学];

学科分类号：

07 ; 0710 ; 09 ;

摘要：

Exposure to hyperoxia, invasive mechanical ventilation, and systemic/local sepsis are important antecedents of postnatal inflammation in the pathogenesis of bronchopulmonary dysplasia (BPD). This review will summarize information obtained from animal (baboon, lamb/sheep, rat and mouse) models that pertain to the specific inflammatory agents and signaling molecules that predispose a premature infant to BPD. Birth Defects Research (Part A) 100:189-201, 2014. (c) 2014 Wiley Periodicals, Inc.

引用

页码：189 / 201

页数：13

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