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- [12] FOS AND JUN BIND COOPERATIVELY TO THE AP-1 SITE - RECONSTITUTION INVITROGENES & DEVELOPMENT, 1988, 2 (12B) : 1687 - 1699RAUSCHER, FJVOULALAS, PJFRANZA, BRCURRAN, T
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- [14] AP-1 (Fos/Jun) transcription factors in hematopoietic differentiation and apoptosis (review)INTERNATIONAL JOURNAL OF ONCOLOGY, 1998, 12 (03) : 685 - 700Liebermann, DAGregory, BHoffman, B
- [15] THE FOS/JUN AP-1 TRANSCRIPTION COMPLEX IS REQUIRED FOR TRANSFORMATION BY ABL ONCOGENESBLOOD, 1994, 84 (10) : A514 - A514RAITANO, ABHAMBUCH, TMSAWYERS, C
- [16] THE C-FOS PROTEIN INTERACTS WITH C-JUN/AP-1 TO STIMULATE TRANSCRIPTION OF AP-1 RESPONSIVE GENESCELL, 1988, 54 (04) : 541 - 552CHIU, RBOYLE, WJMEEK, JSMEAL, THUNTER, TKARIN, M
- [17] MAF AND NRL CAN BIND TO AP-1 SITES AND FORM HETERODIMERS WITH FOS AND JUNONCOGENE, 1994, 9 (03) : 675 - 684KERPPOLA, TKCURRAN, T
- [18] CHOP enhancement of gene transcription by interactions with Jun/Fos AP-1 complex proteinsMOLECULAR AND CELLULAR BIOLOGY, 1999, 19 (11) : 7589 - 7599Ubeda, MVallejo, MHabener, JF
- [19] In vivo CRISPR inactivation of Fos promotes prostate cancer progression by altering the associated AP-1 subunit JunONCOGENE, 2021, 40 (13) : 2437 - 2447Riedel, MariaBerthelsen, Martin F.Cai, HuiqiangHaldrup, JakobBorre, MichaelPaludan, Soren R.Hager, HenrikVendelbo, Mikkel H.Wagner, Erwin F.Bakiri, LatifaThomsen, Martin K.
- [20] In vivo CRISPR inactivation of Fos promotes prostate cancer progression by altering the associated AP-1 subunit JunOncogene, 2021, 40 : 2437 - 2447Maria RiedelMartin F. BerthelsenHuiqiang CaiJakob HaldrupMichael BorreSøren R. PaludanHenrik HagerMikkel H. VendelboErwin F. WagnerLatifa BakiriMartin K. Thomsen