Epigenetics and endoplasmic reticulum in podocytopathy during diabetic nephropathy progression

被引:16
作者
Wang, Xiaokang [1 ]
Zhao, Jingqian [2 ]
Li, Yuanqing [1 ]
Rao, Jiaoyu [1 ]
Xu, Gengrui [1 ]
机构
[1] Guangdong Med Univ, Shenzhen Longhua Dist Cent Hosp, Affiliated Cent Hosp Shenzhen Longhua Dist, Dept Pharm, Shenzhen, Peoples R China
[2] Southern Med Univ, Shenzhen Hosp, Dept Pharm, Shenzhen, Peoples R China
关键词
epigenetics; endoplasmic reticulum; podocyte; diabetic nephropathy; inflammation signaling; UNFOLDED PROTEIN RESPONSE; OXIDATIVE STRESS; URSODEOXYCHOLIC ACID; INSULIN-RESISTANCE; KIDNEY; INJURY; AUTOPHAGY; INFLAMMATION; PODOCYTES; PATHWAY;
D O I
10.3389/fimmu.2022.1090989
中图分类号
R392 [医学免疫学]; Q939.91 [免疫学];
学科分类号
100102 ;
摘要
Proteinuria or nephrotic syndrome are symptoms of podocytopathies, kidney diseases caused by direct or indirect podocyte damage. Human health worldwide is threatened by diabetic nephropathy (DN), the leading cause of end-stage renal disease (ESRD) in the world. DN development and progression are largely dependent on inflammation. The effects of podocyte damage on metabolic disease and inflammatory disorders have been documented. Epigenetic and endoplasmic reticulum (ER) stress are also evident in DN. Targeting inflammation pathway and ER stress in podocytes may be a prospective therapy to prevent the progression of DN. Here, we review the mechanism of epigenetics and ER stress on podocyte inflammation and apoptosis, and discuss the potential amelioration of podocytopathies by regulating epigenetics and ER stress as well as by targeting inflammatory signaling, which provides a theoretical basis for drug development to ameliorate DN.
引用
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页数:16
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