Extrahepatic symptoms in the course of GBV-C/HGV infection

被引：1

作者：

Blanc, P. -L. ^{[1
]}

Boumrazne, R. ^{[1
]}

Sarzier, J. -M. ^{[2
]}

Forel, C. ^{[1
]}

机构：

[1] Ctr Hosp Annonay, Serv Med D, F-07103 Annonay, France

[2] Ctr Transfus Sanguine Rhone Alpes, Annonay, France

来源：

MEDECINE ET MALADIES INFECTIEUSES | 2009年 / 39卷 / 04期

关键词：

GBV-C/HGV; HCV; Hepatitis; HEPATITIS-G VIRUS; TRANSMISSION; DISEASE;

D O I：

10.1016/j.medmal.2008.09.030

中图分类号：

R51 [传染病];

学科分类号：

100401 ;

摘要：

The GBV-C/HGV virus has clearly established transmission modes, mainly blood contamination, and occasionally sexual transmission. It is frequently found among transfused patients, intravenous drug abusers, and hemodialysis patients and often associated with HCV. Its hepatic pathogenicity is very weak, marked by a moderate and transitory cytolysis. Chronic carriage is possible, but does not lead to chronic hepatitis. Carriage can be maintained before the virus disappears. The authors report the case of a patient presenting with pleuropericarditis after a blood transfusion without any other etiology than infection by GBV-C/HGV virus. The possible extrahepatic pathogenicity of the virus is suggested. This hypothesis was rarely put forward. (C) 2008 Elsevier Masson SAS. Tous droits reserves.

引用

页码：264 / 266

页数：3

共 10 条

[1] Acute non-A-E hepatitis in the United States and the role of hepatitis G virus infection [J].

Alter, MJ ;

Gallagher, M ;

Morris, TT ;

Moyer, LA ;

Meeks, EL ;

Krawczynski, K ;

Kim, JP ;

Margolis, HS .

NEW ENGLAND JOURNAL OF MEDICINE, 1997, 336 (11) :741-746

[2] Membranoproliferative glomerulonephritis with subendothelial deposits (type 1) associated with hepatitis G virus infection in a renal transplant recipient [J].

Berthoux, P ;

Laurent, B ;

Cecillon, S ;

Berthoux, F .

AMERICAN JOURNAL OF NEPHROLOGY, 1999, 19 (04) :513-518

[3]

Bertuccio Salvatore Nicola, 2005, Infez Med, V13, P42

[4]

De Renzo A, 2002, HAEMATOLOGICA, V87, P714

[5] STUDIES ON TRANSMISSION OF HUMAN VIRAL HEPATITIS TO MARMOSET MONKEYS .1. TRANSMISSION OF DISEASE SERIAL PASSAGES AND DESCRIPTION OF LIVER LESIONS [J].

DEINHARDT, F ;

HOLMES, AW ;

CAPPS, RB ;

POPPER, H .

JOURNAL OF EXPERIMENTAL MEDICINE, 1967, 125 (04) :673-+

[6]

DELAUGERRE C, 1998, LETT INFECT, V13, P434

[7] Evidence for probable sexual transmission of the hepatitis G virus [J].

Frey, SE ;

Homan, SM ;

Sokol-Anderson, M ;

Cayco, MT ;

Cortorreal, P ;

Musial, CE ;

Di Bisceglie, A .

CLINICAL INFECTIOUS DISEASES, 2002, 34 (08) :1033-1038

[8] GBV-C/HGV is not the major cause of autoimmune hepatitis [J].

Heringlake, S ;

Tillmann, HL ;

CordesTemme, P ;

Trautwein, C ;

Hunsmann, G ;

Manns, MP .

JOURNAL OF HEPATOLOGY, 1996, 25 (06) :980-984

[9] Hepatitis G virus biology, epidemiology, and clinical manifestations: Implications for blood safety [J].

Kleinman, S .

TRANSFUSION MEDICINE REVIEWS, 2001, 15 (03) :201-212

[10] DETECTION OF THE GBV-C HEPATITIS-VIRUS GENOME IN SERUM FROM PATIENTS WITH FULMINANT-HEPATITIS OF UNKNOWN ETIOLOGY [J].

YOSHIBA, M ;

OKAMOTO, H ;

MISHIRO, S .

LANCET, 1995, 346 (8983) :1131-1132

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