Acquired epidermodysplasia verruciformis

被引：85

作者：

Rogers, Heather D. ^{[1
]}

MacGregor, Jennifer L. ^{[1
]}

Nord, Kristin M. ^{[1
]}

Tyring, Stephen ^{[3
]}

Rady, Peter ^{[3
]}

Engler, Danielle E. ^{[1
]}

Grossman, Marc E. ^{[1
,2
]}

机构：

[1] Columbia Univ, Med Ctr, Dept Dermatol, New York, NY USA

[2] Columbia Univ, Med Ctr, Dermatol Consultat Serv, New York, NY USA

[3] Univ Texas Houston, Hlth Sci Ctr, Dept Dermatol, Houston, TX USA

来源：

JOURNAL OF THE AMERICAN ACADEMY OF DERMATOLOGY | 2009年 / 60卷 / 02期

关键词：

HUMAN-IMMUNODEFICIENCY-VIRUS; ACTIVE ANTIRETROVIRAL THERAPY; HUMAN-PAPILLOMAVIRUS TYPE-5; HIV-INFECTION; PATIENT; ERUPTION; EXPRESSION; RECIPIENTS; DISEASE; CANCERS;

D O I：

10.1016/j.jaad.2008.08.035

中图分类号：

R75 [皮肤病学与性病学];

学科分类号：

100206 ;

摘要：

Epidermodysplasia verruciformis (EV) is a rare autosomal recessive genodermatosis with an increased susceptibility to Specific human papillomavirus (HPV) genotypes. Classically, this viral infection leads to the development of: tinea versicolor-like macules on the trunk, neck, arms, and face during Childhood, and over time, these lesions can progress to squamous Cell carcinoma. More recently, an EV-like syndrome has been described in patients with impaired cell-mediated immunity. We describe two cases of EV-like syndrome in HIV-positive patients, review all previously reported cases of EV in patients with impaired cell-mediated immunity, introduce the term "acquired epidermodysplasia verruciformis" to describe EV developing in the immunocompromised host and examine the limited treatment options for these patients. (J Am Acad Dermatol 2009-160:315-20.)

引用

页码：315 / 320

页数：6

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