NFATs are good for your cartilage!

被引：6

作者：

Beier, F. ^{[1
]}

机构：

[1] Univ Western Ontario, Dept Physiol & Pharmacol, Schulich Sch Med & Dent, London, ON N6A 5C1, Canada

来源：

OSTEOARTHRITIS AND CARTILAGE | 2014年 / 22卷 / 07期

基金：

加拿大健康研究院;

关键词：

Osteoarthritis; NFAT; Mouse models; Articular cartilage; TRANSCRIPTION FACTOR ERG; NUCLEAR FACTOR; OSTEOARTHRITIS; CHONDROCYTES; EXPRESSION; CELLS; BONE; CHONDROGENESIS; CALCINEURIN; ACTIVATION;

D O I：

10.1016/j.joca.2014.04.011

中图分类号：

R826.8 [整形外科学]; R782.2 [口腔颌面部整形外科学]; R726.2 [小儿整形外科学]; R62 [整形外科学（修复外科学）];

学科分类号：

摘要：

引用

页码：893 / 895

页数：3

共 20 条

[1] WNT5A Regulates Chondrocyte Differentiation through Differential Use of the CaN/NFAT and IKK/NF-κB Pathways [J].

Bradley, Elizabeth W. ;

Drissi, M. Hicham .

MOLECULAR ENDOCRINOLOGY, 2010, 24 (08) :1581-1593

[2] Deletion of Glycogen Synthase Kinase-3β in Cartilage Results in Up-Regulation of Glycogen Synthase Kinase-3α Protein Expression [J].

Gillespie, J. R. ;

Ulici, V. ;

Dupuis, H. ;

Higgs, A. ;

DiMattia, A. ;

Patel, S. ;

Woodgett, J. R. ;

Beier, F. .

ENDOCRINOLOGY, 2011, 152 (05) :1755-1766

[3] NFATc1 and NFATc2 repress spontaneous osteoarthritis [J].

Greenblatt, Matthew B. ;

Ritter, Susan Y. ;

Wright, John ;

Tsang, Kelly ;

Hu, Dorothy ;

Glimcher, Laurie H. ;

Aliprantis, Antonios O. .

PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 2013, 110 (49) :19914-19919

[4] Subchondral bone and osteoarthritis: biological and cellular aspects [J].

Henrotin, Y. ;

Pesesse, L. ;

Sanchez, C. .

OSTEOPOROSIS INTERNATIONAL, 2012, 23 :S847-S851

[5] Transcription factor ERG variants and functional diversification of chondrocytes during limb long bone development [J].

Iwamoto, M ;

Higuchi, Y ;

Koyama, E ;

Enomoto-Iwamoto, M ;

Kurisu, K ;

Yeh, H ;

Abrams, WR ;

Rosenbloom, J ;

Pacifici, M .

JOURNAL OF CELL BIOLOGY, 2000, 150 (01) :27-39

[6] Transcription factor ERG and joint and articular cartilage formation during mouse limb and spine skeletogenesis [J].

Iwamoto, Masahiro ;

Tamamura, Yoshihiro ;

Koyama, Eiki ;

Komori, Toshihisa ;

Takeshita, Nobuo ;

Williams, Julie A. ;

Nakamura, Takashi ;

Enomoto-Iwamoto, Motomi ;

Pacifici, Maurizio .

DEVELOPMENTAL BIOLOGY, 2007, 305 (01) :40-51

[7] Osteoarthritis: A disease of the joint as an organ [J].

Loeser, Richard F. ;

Goldring, Steven R. ;

Scanzello, Carla R. ;

Goldring, Mary B. .

ARTHRITIS AND RHEUMATISM, 2012, 64 (06) :1697-1707

[8] Inhibition of Gsk3β in cartilage induces osteoarthritic features through activation of the canonical Wnt signaling pathway [J].

Miclea, R. L. ;

Siebelt, M. ;

Finos, L. ;

Goeman, J. J. ;

Lowik, C. W. G. M. ;

Oostdijk, W. ;

Weinans, H. ;

Wit, J. M. ;

Robanus-Maandag, E. C. ;

Karperien, M. .

OSTEOARTHRITIS AND CARTILAGE, 2011, 19 (11) :1363-1372

[9] Cartilage biology in osteoarthritis-lessons from developmental biology [J].

Pitsillides, Andrew A. ;

Beier, Frank .

NATURE REVIEWS RHEUMATOLOGY, 2011, 7 (11) :654-663

[10] The nuclear factor of activated T cells (NFAT) transcription factor NFATp (NFATc2) is a repressor of chondrogenesis [J].

Ranger, AM ;

Gerstenfeld, LC ;

Wang, JX ;

Kon, T ;

Bae, HS ;

Gravallese, EM ;

Glimcher, MJ ;

Glimcher, LH .

JOURNAL OF EXPERIMENTAL MEDICINE, 2000, 191 (01) :9-21

← 1 2 →