Protease-activated receptors and prostaglandins in inflammatory lung disease

被引:51
作者
Peters, Terence [1 ]
Henry, Peter J. [1 ]
机构
[1] Univ Western Australia, Sch Med & Pharmacol, Nedlands, WA 6009, Australia
基金
英国医学研究理事会;
关键词
protease-activated receptors; prostaglandins; airway inflammation; asthma; allergy; fibrosis; AIRWAY SMOOTH-MUSCLE; REGULATORY T-CELLS; CILIARY BEAT FREQUENCY; GROWTH-FACTOR-BETA; PEPDUCIN-BASED INTERVENTION; BRONCHIAL EPITHELIAL-CELLS; INDUCED PULMONARY-FIBROSIS; CAMP-ELEVATING AGENTS; NECROSIS-FACTOR-ALPHA; GUINEA-PIG TRACHEA;
D O I
10.1111/j.1476-5381.2009.00449.x
中图分类号
R9 [药学];
学科分类号
1007 ;
摘要
This article is part of a themed issue on Mediators and Receptors in the Resolution of Inflammation. To view this issue visit http://www3.interscience.wiley.com/journal/121548564/issueyear?year=2009.
引用
收藏
页码:1017 / 1033
页数:17
相关论文
共 292 条
[1]   Protease-activated receptor-2 peptides activate neurokinin-1 receptors in the mouse isolated trachea [J].
Abey, HT ;
Fairlie, DP ;
Moffatt, JD ;
Balzary, RW ;
Cocks, TM .
JOURNAL OF PHARMACOLOGY AND EXPERIMENTAL THERAPEUTICS, 2006, 317 (02) :598-605
[2]   New targets for drug development in asthma [J].
Adcock, Ian M. ;
Caramori, Gaetano ;
Chung, K. Fan .
LANCET, 2008, 372 (9643) :1073-1087
[3]   Inhibition of cellular action of thrombin by N3-cyclopropyl-7-{[4-(1-methylethyl)phenyl]methyl}-7H-pyrrolo[3,2-f]quinazoline-1,3-diamine (SCH 79797), a nonpeptide thrombin receptor antagonist [J].
Ahn, HS ;
Foster, C ;
Boykow, G ;
Stamford, A ;
Manna, M ;
Graziano, M .
BIOCHEMICAL PHARMACOLOGY, 2000, 60 (10) :1425-1434
[4]   Guide to receptors and channels (GRAC), 3rd edition [J].
Alexander, Stephen P. H. ;
Mathie, Alistair ;
Peters, John A. .
BRITISH JOURNAL OF PHARMACOLOGY, 2008, 153 :S1-S209
[5]   Frequent acetaminophen use and allergic diseases: Is the association clear? [J].
Allmers, H .
JOURNAL OF ALLERGY AND CLINICAL IMMUNOLOGY, 2005, 116 (04) :859-862
[6]   Tumor necrosis factor-α-induced secretion of RANTES and interleukin-6 from human airway smooth-muscle cells -: Modulation by cyclic adenosine monophosphate [J].
Ammit, AJ ;
Hoffman, RK ;
Amrani, Y ;
Lazaar, AL ;
Hay, DWP ;
Torphy, TJ ;
Penn, RB ;
Panettieri, RA .
AMERICAN JOURNAL OF RESPIRATORY CELL AND MOLECULAR BIOLOGY, 2000, 23 (06) :794-802
[7]   Endotyping asthma: new insights into key pathogenic mechanisms in a complex, heterogeneous disease [J].
Anderson, Gary P. .
LANCET, 2008, 372 (9643) :1107-1119
[8]   Proteinase-activated receptor 4 stimulation-induced epithelial-mesenchymal transition in alveolar epithelial cells [J].
Ando, Seijitsu ;
Otani, Hitomi ;
Yagi, Yasuhiro ;
Kawai, Kenzo ;
Araki, Hiromasa ;
Fukuhara, Shirou ;
Inagaki, Chiyoko .
RESPIRATORY RESEARCH, 2007, 8 (1)
[9]   INVESTIGATION OF THE INHIBITORY EFFECTS OF PGE(2) AND SELECTIVE EP AGONISTS ON CHEMOTAXIS OF HUMAN NEUTROPHILS [J].
ARMSTRONG, RA .
BRITISH JOURNAL OF PHARMACOLOGY, 1995, 116 (07) :2903-2908
[10]   Prostaglandin E2 inhibits alveolar macrophage phagocytosis through an E-prostanoid 2 receptor-mediated increase in intracellular cyclic AMP [J].
Aronoff, DM ;
Canetti, C ;
Peters-Golden, M .
JOURNAL OF IMMUNOLOGY, 2004, 173 (01) :559-565